Ok now I wait...I've contacted my rheum who said she did extensive work and nothing was there
the allergist found this for me
so I told them they did check everything except my igg4
and it's scary what I'm finding online...
but could be why I'm way I am
i Lao had biopsy on stomach because I have gastritis some how
on dairy gluten wheat free diet
maybe stress..
they are looking for cancer and h pyloria
im also awaiting extensive blood work on horomones
as my lh hormone was really high which promoted them to do MRI of brain
and nothing was shown...
but ive yet to revive the results I'm going to ask for tjem
also need my medical records
because I was told years ago I had thyroiditis from tsh being 5.76 and ultra sound showed it dying inflamed
and was put on levothyroxine and my tsh went down on .50 and my weight plummeted from 150-119 in two months
so I'm still curious how my thyroid healed and I haven't been on levo in years lol
also I have every symptom of sjorgens
dry eye dry nose dry skin dry scalp
mouth really dry when waking up
not so bad if I brush teeth more and drink water
im tired a lot since this diet
my skin resembles Rosacea or lupus like
I avoid sun
and I have insomnia and average 4 hours a night
cold hands and feet
and anxiety depression OCD anger from all of this...I'll keep u like informed
this igg4 could be big
my liver enzymes use to be high as well
and they never did biopsy when they said they would....

anyoned dick growth been stunted on this stuff? can you all not grow beards?

My penis at the time on accutane I believe shrunk due to dryness
but I'm comfortable with the size
just wonder if it would have been even bigger had I not been so dried up
coocnut oil on penis before bed helps dryness ...beard growth ehh I can't really grow one that good
I am however growing hair up to eyes and close to t zone though
laser hair removal didn't help

6 hours ago, Nemesisbrady said:

Ok now I wait...I've contacted my rheum who said she did extensive work and nothing was there
the allergist found this for me
so I told them they did check everything except my igg4
and it's scary what I'm finding online...
but could be why I'm way I am
i Lao had biopsy on stomach because I have gastritis some how
on dairy gluten wheat free diet
maybe stress..
they are looking for cancer and h pyloria
im also awaiting extensive blood work on horomones
as my lh hormone was really high which promoted them to do MRI of brain
and nothing was shown...
but ive yet to revive the results I'm going to ask for tjem
also need my medical records
because I was told years ago I had thyroiditis from tsh being 5.76 and ultra sound showed it dying inflamed
and was put on levothyroxine and my tsh went down on .50 and my weight plummeted from 150-119 in two months
so I'm still curious how my thyroid healed and I haven't been on levo in years lol
also I have every symptom of sjorgens
dry eye dry nose dry skin dry scalp
mouth really dry when waking up
not so bad if I brush teeth more and drink water
im tired a lot since this diet
my skin resembles Rosacea or lupus like
I avoid sun
and I have insomnia and average 4 hours a night
cold hands and feet
and anxiety depression OCD anger from all of this...I'll keep u like informed
this igg4 could be big
my liver enzymes use to be high as well
and they never did biopsy when they said they would....

Although not validated yet in clinical trials, the common induction regime isprednisolone3040mg per day for 24 weeks, then gradually tapered over 3 to 6 months. Recurrences during or after tapering of glucocorticoids are frequent however.Steroid-sparing immunosuppressive agentsmight be considered, depending on local availability of these drugs, for use in combination with glucocorticoids from the start of treatment. Steroid-sparing agents that have been used includerituximab,azathioprine,methotrexate, andcyclophosphamide, although trials are needed to ascertain the effectiveness of each drug in IgG4-RD.

Have my neurologist appointment coming up. Like I said, they can't avoid the fact that my vocal cord is partially paralyzed. Will keep y'all updated. Autoimmune causing peripheral neuropathy is very possible. Mostly reversible when the underlying cause is treated which makes me feel better.

Patented POISONS = isotretinoin. I always wondered why some derms won't use it in their practice NOW I know why this is one of those drugs your mother warned you about.

Perhaps your parent took this at one point in their lives and remembers the devastating consequences of it later in life.

https://rxisk.org/accutane-30-years-of-trading-our-sex-lives-for-clear-skin/

Walden rev thank u
naybe this is why some ppl do good on plaquenil for flushing

My neurologist prescribed me an anti depressant to treat what he calls "migraines" and a couple of weeks, I will say the head pressure has subsided and the muscle aches aren't bad when I first wake up in the morning.

I I know it's a short term solution because I already feel myself becoming emotionaly numb, but I guess it's a temporary trade off.

49 minutes ago, Jorcruz24 said:

My neurologist prescribed me an anti depressant to treat what he calls "migraines" and a couple of weeks, I will say the head pressure has subsided and the muscle aches aren't bad when I first wake up in the morning.

I I know it's a short term solution because I already feel myself becoming emotionaly numb, but I guess it's a temporary trade off.

Must be Endep?

9 hours ago, mikez said:

Must be Endep?

Amitriptyline 10 mg daily

so so far head pressure is down and I muscle pain is gone. I'm crossing my fingers that this lasts.

On 5/14/2017 at 10:52 PM, Chris16 said:

I agree Iodine and LDN are very powerful to heal us. Make sure if you're taking iodine that you take the co factors. I'm up to 20 mg and starting to see results.

Can you keep us up-dated please,

21 hours ago, mikez said:

Must be Endep?

Why would it necessary be Endep can I ask??

12 hours ago, Jorcruz24 said:

Amitriptyline 10 mg daily

so so far head pressure is down and I muscle pain is gone. I'm crossing my fingers that this lasts.

Excellent!!!

Keep us updated as you progress. Any side effects yet?

Also, can you elaborate on the consultation you had with your doctor?? Why did he/she specifically prescribe Amitriptyline?
Did you discuss Accutane and that prompted them to recommend this or was it just a stab in the dark from them thinking this might help you?

I ask as I too suffer head pressure/depression...and can't get relief naturally with anything, I'm considering going back on something like this if it helps!!

3 hours ago, TrueJustice said:

Why would it necessary be Endep can I ask??

Its the only anti depressant I can think of that is also approved for migraines as well.

32 minutes ago, mikez said:

Its the only anti depressant I can think of that is also approved for migraines as well.

I didn't know that. I was on Endep for a brief period about 13 years ago, if I'm not mistaken I took it for improving sleep!?
im sure it was Endep

It worked but once you come of it, it was back to square one for me.....

I have no desire to go permanently on a prescribed drug, others may disagree...except for Pariot - this works for my reflux, if I miss taking it for say 2 days, back comes the reflux!!!!

27 minutes ago, TrueJustice said:

I didn't know that. I was on Endep for a brief period about 13 years ago, if I'm not mistaken I took it for improving sleep!?
im sure it was Endep

It worked but once you come of it, it was back to square one for me.....

I have no desire to go permanently on a prescribed drug, others may disagree...except for Pariot - this works for my reflux, if I miss taking it for say 2 days, back comes the reflux!!!!

Yes , it's a potent anti histamine, so also used for sleep, but that is off label, as opposed to the headache indication.

I agree in some aspects, if its many drugs and high doses, but if I can feel a lot better with anything , I'm open to it.

I'm trying LDN now, which is big in the chronic disease forums, and that's also a prescribed drug technically, so I always keep an open mind.

53 minutes ago, mikez said:

Yes , it's a potent anti histamine, so also used for sleep, but that is off label, as opposed to the headache indication.

I agree in some aspects, if its many drugs and high doses, but if I can feel a lot better with anything , I'm open to it.

I'm trying LDN now, which is big in the chronic disease forums, and that's also a prescribed drug technically, so I always keep an open mind.

Fair enough, I hope it works for you, I could very well be taking it myself in the future - plenty of people are certainly talking about LDN.

As I contemplate the 20 years of this bullshit, I can't help lately to feel incredibly disappointed in the body's ability to heal itself or lack thereof....
Why do health experts always say this shit, why hasn't it happened?? The body wants to heal itself, yeah right when?

Its like that article on previous page saying "30 years of sexual side effects" from Accutane, Who came up with that as an indicator?? So does that mean after 30 years we finally become healed?
why not 32 or 35 years.....bullshit....who writes this stuff, if you're going to say that in an article, provide an interview with someone who's survived those 30 years and is now fine, then I'll believe it!!

Hey everyone, this is just a 5 months update from this post. So far, I think that this is method of tackling the side effects of accutane is very effective. It isn't instant, but neither is it expensive or risky in the slightest. Even if you had no health problems doing these things would only improve your health, and most, if not all, of these things you could buy at the supplement aisle of your supermarket, maybe for creatine you may have to go to a supplement store, but I have seen them in supermarkets too, usually just a bit dearer. 

Currently I feel I am pretty much 100%, and maybe I am lucky, but I just wanted to let you know it has worked for me, and the post below explains why it should work in greater detail

On 13/03/2017 at 2:51 PM, Fchawk said:

Long Post, but highly recommend you read it all

 

What do we know about accutane? Well certainly the people that make it claim to know nothing. All they claim to know is that it causes birth defects, and it causes acne to go away. They also claim it causes no lasting side effects, which is obviously incorrect. However, that is because not everyone suffers the same side effects. The reason for this, is because it inhibits neurogenesis and causes hippocampal atrophy, which causes changes in brain metabolism similar to those caused by traumatic brain injuries, aka, varying degrees of brain damage. The after effects of suffering brain trauma can be severe, especially if left unmanaged, but there are many things you can do to ensure you get very close to a 100% recovery, which in in the second half of the post. Skip down if you don't need to see the research behind the advice

 

http://www.ncbi.nlm.nih.gov/pubmed/25689814

"A 25-question survey was emailed to 7,013 dermatologists included in a proprietary database (MBD, Inc.) and anonymous responses were collected. 591 board-certified dermatologists participated. Thirty-seven percent of the responding dermatologists believe that isotretinoin may cause psychiatric disturbances. Dermatologists' opinions on this relationship did not significantly impact prescription practices in patients with history of depression (P=0.056) or in patients being treated with an antidepressant (P=0.118)."

 

http://www.ncbi.nlm.nih.gov/pubmed/15863802
 

"Results: Isotretinoin but not antibiotic treatment was associated with decreased brain metabolism in the orbitofrontal cortex (-21% change versus 2% change for antibiotic), a brain area known to mediate symptoms of depression. 
Conclusion: This study suggests that isotretinoin treatment is associated with changes in brain functioning."

 

œA 4-month treatment trial with isotretinoin was associated with a decrease in brain functioning in the orbito-frontal cortex, a brain region implicated in depression.

 

http://www.ncbi.nlm.nih.gov/pubmed/15251924

œWe now show, in a mouse model, that endogenous RA generated by synthetic enzymes in the meninges acts on hippocampal granule neurons, and chronic (3-week) exposure to a clinical dose of 13-cis RA may result in hippocampal cell loss.

 

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC387382/

"This report demonstrates that a clinical dose (1 mg/kg/day) of 13-cis-RA in mice significantly reduces cell proliferation in the hippocampus and the subventricular zone, suppresses hippocampal neurogenesis, and severely disrupts capacity to learn a spatial radial maze task. The results demonstrate that the regions of the adult brain where cell proliferation is ongoing are highly sensitive to disruption by a clinical dose of 13-cis-RA."

 

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3276716/#R173

"Increased concentrations of homocysteine have also been associated with attacks of violent anger. Isotretinoin administration to human subjects was shown to be associated with increased concentrations of homocysteine, as well as decreases in 5-methyl-tetrahydrofolate, providing a potential metabolic mechanism by which isotretinoin may promote depression."

"In the case of patients reported to the Norwegian Medicines Agency, single photon emission computed tomography (SPECT) of the brain was performed in 15 cases who reported lasting neurological symptoms. Altered brain function was seen in all cases involving altered or reduced frontal lobe blood flow. Ten of these patients were evaluated to have organic brain damage."

 

https://www.ncbi.nlm.nih.gov/pubmed/20708044

"13-Cis-retinoic acid (13-cis-RA) causes depression-related behavior in mice. Hypothalamic dysregulation has been implicated in clinical depression. In fact, apoptosis of hypothalamic neurons may lead to depression after myocardial infarction. . . .We hypothesize that the ability of 13-cis-RA to decrease hypothalamic cell number may contribute to the increased depression-related behaviors observed in mice."

 

Now that we have established that Accutane causes hippocampal atrophy and varying degrees of brain damahe, what are the consequences?

 

Traumatic brain injury: a disease process, not an event.

https://www.ncbi.nlm.nih.gov/m/pubmed/20504161/

œTraumatic brain injury (TBI) is seen by the insurance industry and many health care providers as an "event." Once treated and provided with a brief period of rehabilitation, the perception exists that patients with a TBI require little further treatment and face no lasting effects on the central nervous system or other organ systems. In fact, TBI is a chronic disease process, one that fits the World Health Organization definition as having one or more of the following characteristics: it is permanent, caused by non-reversible pathological alterations, requires special training of the patient for rehabilitation, and/or may require a long period of observation, supervision, or care. TBI increases long-term mortality and reduces life expectancy. It is associated with increased incidences of seizures, sleep disorders, neurodegenerative diseases, neuroendocrine dysregulation, and psychiatric diseases, as well as non-neurological disorders such as sexual dysfunction, bladder and bowel incontinence, and systemic metabolic dysregulation that may arise and/or persist for months to years post-injury. The purpose of this article is to encourage the classification of TBI as the beginning of an ongoing, perhaps lifelong process, that impacts multiple organ systems and may be disease causative and accelerative. Our intent is not to discourage patients with TBI or their families and caregivers, but rather to emphasize that TBI should be managed as a chronic disease and defined as such by health care and insurance providers. Furthermore, if the chronic nature of TBI is recognized by government and private funding agencies, research can be directed at discovering therapies that may interrupt the disease processes months or even years after the initiating event.

 

Currently there is no acknowledgement of this from anywhere, which is why mental illness is becoming an epidemic. In a few decades though I think this will become mainstream knowledge

 

https://www.ncbi.nlm.nih.gov/pubmed/16425236

œThe hippocampus is one of several limbic brain structures implicated in the pathophysiology and treatment of mood disorders. Preclinical and clinical studies demonstrate that stress and depression lead to reductions of the total volume of this structure and atrophy and loss of neurons in the adult hippocampus. One of the cellular mechanisms that could account for alterations of hippocampal structure as well as function is the regulation of adult neurogenesis. Stress exerts a profound effect on neurogenesis, leading to a rapid and prolonged decrease in the rate of cell proliferation in the adult hippocampus. In contrast, chronic antidepressant treatment up-regulates hippocampal neurogenesis, and could thereby block or reverse the atrophy and damage caused by stress. Recent studies also demonstrate that neurogenesis is required for the actions of antidepressants in behavioral models of depression. This review discusses the literature that has lead to a neurogenic hypothesis of depression and antidepressant action, as well as the molecular and cellular mechanisms that underlie the regulation of adult neurogenesis by stress and antidepressant treatment.

 

In this we see that accutane, in many ways, affects us like a chronic bout of stress. It is also why people who use antidepressants feel better, and why you often find people recommending SSRIs to treat accutane™s sides. That is because they DO help.

Now we established that hippocampal atrophy may be the cause of many of our symptoms, and that the way antidepressants work is by stimulating neurogenesis, here is how you can improve your recovery naturally, though if you wish to use antidepressants as well that fine as well

 

 

Nutritional treatment for acute and chronic traumatic brain injury patients.

https://www.ncbi.nlm.nih.gov/m/pubmed/24844176/?i=6&from=/24605947/related

"omega 3 fats, vitamin D, N-Acetylcysteine, branched chain amino acids, zinc, alpha-lipoic acid, magnesium, taurine, coenzyme Q10, and many phytonutrients may be helpful in the recovery from a a TBI"

 

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3705194/

Mindfulness Meditation can stimulate hippocampal brain cell growth. A smaller hippocampus is correlated with a poorer recovery from TBIs, in the case of war veterans suffering PTSD at least.

 

http://www.ncbi.nlm.nih.gov/m/pubmed/11079535/

Study supporting Creatine consumption as one of the top supplements for recovering from a TBI, and the one below supports Taurine use as well.

http://www.ncbi.nlm.nih.gov/m/pubmed/27156064/

 

Long-term effects of a ketogenic diet in obese patients

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2716748/

 

"Beneficial changes in the brain energy profile have been observed in subjects who are on a ketogenic diet (28). This is a significant observation because cerebral hypometabolism is a characteristic feature of those who suffer from depression or mania"

 

Currently I  doing:

Creatine - increases Dihydrotestosterone (DHT) and testosterone. Increases muscle power and ALSO improves neuroplasticity

Fish Oil - improves joint pain, helps heart disease, and ALSO improves neuroplasticity

Zinc - increases levels of male hormones and ALSO improves neuroplasticity

Magnesium - helps with chronic pain, fatigue and insomnia and ALSO neuroplasticity

Vitamin D: Improves bone health, physical fitness, and ALSO improves neuroplasticity
CoQ10: Improves cardiovascular fitness and heart health, and ALSO improves neuroplasticity

Multivitamin - makes me less likely to be malnourished.

I try to do meditation regularly, but I don't prioritise it enough... Though it helps with anxiety and stress, as well as neuroplasticity, and i plan to add it into my daily routine.  

I feel much better than I have in years. I think for the last two months I have woken with morning wood 95% of the time, social anxiety is limited, confidence in my abilities in much higher, and motivation to reach my goals, and also willingness to do the work to reach them, is also one of the main things I have noticed. Before I did the work still expecting to fail, while now I do the work and I expect success, which makes me actually enjoy the work, and that™s just one example. My life and my future doesn™t seem so hopeless, there is plenty to celebrate and I™m sure plenty I will celebrate in the future

 

Things I plan to do:

Taurine: Helps body avoid hypervitaminosis A, improves eyesight, digestion, heart health and ALSO improves neuroplasticity

Ketogenic Diet: Improves body composition, can help ED, and ALSO improves neuroplasticity

I try to do meditation regularly, but I don't prioritise it enough... Though it helps with anxiety and stress, as well as neuroplasticity, and i plan to add it into my daily routine.  

 

Things that might help, but are on the riskier side and I am unlikely to attempt myself, but possibly would help

 

Antidepressants increase human hippocampal neurogenesis by activating the glucocorticoid receptor

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3121947/

 

The regulation of adult rodent hippocampal neurogenesis by deep brain stimulation.

https://www.ncbi.nlm.nih.gov/pubmed/18173322

œHigh-frequency stimulation of the AN increases the hippocampal neurogenesis and restores experimentally suppressed neurogenesis. Interventions that increase hippocampal neurogenesis have been associated with enhanced behavioral performance. In this context, it may be possible to use electrical stimulation to treat conditions associated with impairment of hippocampal function.

 

Stimulation of entorhinal cortex promotes adult neurogenesis and facilitates spatial memory.

https://www.ncbi.nlm.nih.gov/pubmed/21940440

œDeep brain stimulation (DBS) is an established therapeutic modality for the treatment of movement disorders and an emerging therapeutic approach for the treatment of disorders of mood and thought. For example, recently we have shown that DBS of the fornix may ameliorate cognitive decline associated with dementia. However, like other applications of DBS, the mechanisms mediating these clinical effects are unknown. As DBS modulates neurophysiological activity in targeted brain regions, DBS might influence cognitive function via activity-dependent regulation of hippocampal neurogenesis. Using stimulation parameters analogous to clinical high-frequency DBS, here we addressed this question in mice. We found that acute stimulation of the entorhinal cortex (EC) transiently promoted proliferation in the dentate gyrus (DG). Cells generated as a consequence of stimulation differentiated into neurons, survived for at least several weeks, and acquired normal dentate granule cell (DGC) morphology. Importantly, stimulation-induced promotion of neurogenesis was limited to the DG and not associated with changes in apoptotic cell death. Using immunohistochemical approaches, we found that, once sufficiently mature, these stimulation-induced neurons integrated into hippocampal circuits supporting water-maze memory. Finally, formation of water-maze memory was facilitated 6 weeks (but not 1 week) after bilateral stimulation of the EC. The delay-dependent nature of these effects matches the maturation-dependent integration of adult-generated DGCs into dentate circuits supporting water-maze memory. Furthermore, because the beneficial effects of EC stimulation were prevented by blocking neurogenesis, this suggests a causal relationship between stimulation-induced promotion of adult neurogenesis and enhanced spatial memory.

 

Nootropic agents stimulate neurogenesis.

https://www.ncbi.nlm.nih.gov/pubmed/19441945

 

Electrical Stimulation Elicits Neural Stem Cells Activation: New Perspectives in CNS Repair

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4610200/

 

Acupuncture stimulation induces neurogenesis in adult brain.

https://www.ncbi.nlm.nih.gov/pubmed/24215918

 

Hippocampal Neurogenesis and Antidepressive Therapy: Shocking Relations

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4055571/
"A strong enhancement of neurogenesis has been observed in various species following experimental ECS treatments [20, 21]. Several studies indicated a close relation between hippocampal function and mood regulation. The observation of an antidepressive-like effect and an upregulation of hippocampal cell proliferation upon experimental ECS raised speculations on the participation of neurogenesis in the antidepressive mode of action. However, evidence for a direct participation of neurogenesis in antidepressive mechanisms still remains to be convincingly demonstrated [17].

Systematic review of effect of coenzyme Q10 in physical exercise, hypertension and heart failure.

https://www.ncbi.nlm.nih.gov/pubmed/14695924

COENZYME Q10 IN PHYSICAL EXERCISE. We identified eleven studies in which CoQ10 was tested for an effect on exercise capacity, six showed a modest improvement in exercise capacity with CoQ10 supplementation but five showed no effect. CoQ10 IN HYPERTENSION. We identified eight published trials of CoQ10 in hypertension. Altogether in the eight studies the mean decrease in systolic blood pressure was 16 mm Hg and in diastolic blood pressure, 10 mm Hg. Being devoid of significant side effects CoQ10 may have a role as an adjunct or alternative to conventional agents in the treatment of hypertension. CoQ10 IN HEART FAILURE. We performed a randomised double blind placebo-controlled pilot trial of CoQ10 therapy in 35 patients with heart failure. Over 3 months, in the CoQ10 patients but not in the placebo patients there were significant improvements in symptom class and a trend towards improvements in exercise time.

 

Smaller hippocampal volume predicts pathologic vulnerability to psychological trauma
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2819093/

 

œIn animals, exposure to severe stress can damage the hippocampus. Recent human studies show smaller hippocampal volume in individuals with the stress-related psychiatric condition posttraumatic stress disorder (PTSD). Does this represent the neurotoxic effect of trauma, or is smaller hippocampal volume a pre-existing condition that renders the brain more vulnerable to the development of pathological stress responses? In monozygotic twins discordant for trauma exposure, we found evidence that smaller hippocampi indeed constitute a risk factor for the development of stress-related psychopathology. Disorder severity in PTSD patients who were exposed to trauma was negatively correlated with the hippocampal volume of both the patients and the patients™ trauma-unexposed identical co-twin. Furthermore, severe PTSD twin pairs”both the trauma-exposed and unexposed members”had significantly smaller hippocampi than non-PTSD pairs.

 

here is another interesting study about recovering from a TBI, it's basically like the worse the patient thinks his recovery will be, the worse it will be

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2077969/

The reason for this may be the worse the TBI is, the less likely the patient is optimistic about his recovery, or the worse his mental state before the injury happened the worse his recovery will be, rather than being optimistic improves outcomes

 

This is pretty much all the information needed for taking this approach to curing Post-Accutane Syndrome in one place. Obviously I would have missed things, and thats why I want everyone to discuss and suggest things that I have missed, and while I am certain that this will wipe away many of the sides, there are other mechanisms in which accutane acted, and while this counters one way it wrecked havoc in our lives, any ideas of the things we may do to address its other issues will always be appreciated

Love you all, don™t give up, we will continue getting better together!

All in all, I think that I am having a lot of success with this, and encourage any interested to try it as well. Pretty much all the necessary research is up above, and I think that this is the best way to make a full recovery. Good Luck!

I am finding success with coffee, pregnenolone, and Silymarin (Milk Thistle). I am getting a lot of sleep, actually probably more than one should (10+ hrs), maybe that's good, in our case, for healing? And I feel generally good. Sometimes I get blasts of dopamine throughout the day. Wake up some days with morning erections (even though i have proven low T). I start TRT next week and hyperbaric oxygen therapy. Fingers crossed.

Yes, I wondered if a ketogenic diet would be beneficial for us? I think though I prefer a fruit based/plant based diet, with expensive cuts of meat and fish once or twice a week.

Quick update on my situation:
finished 5 day water fast one week ago. Result: better deeper sleep, more appetite since finished, more energy (ups & downs), better sexual conditions, no brain fog( was not a big problem before) overall maybe 10% recovered still feel very bad.
Will start a juice fast 1. September 2 weeks. Have buyed finasterid and will start on the lowest dosage after juice fast. Everybody here do not give up we will do this.

Waow . It's been a long time since we haven't seen such great results ! The three post above are more than encouraging ! Congrats !
moreover what you did guys were exactly in my mind and I was considering to try all you mentioned , as well as the CDNUTS protocol
I myself have reduced carbs intake and feel a lot better . I noticed that when I eat carbs , especially during the day, I crash right after . A lot of people reported it too. Nothing dramatically improved but my time will come as well. In a few months ( around May of next year ) I'll take a gap year or two focusing only on the recovery , currently doing the necessary about my study and money to make it possible .
Again, congrats for your results and thanks for sharing . Godspeed

Just had some bloods repeated, and again Inorganic phosphate level is low, quite a bit below range.

This could indicate low vitamin D, but this would seem unlikely as I supplement 10,000ui every 2 days, and get plenty of sun (had this tested, waiting results but in the past it's been in range, although low end)

Low inorganic phosphate could also indicate Wilson's disease, and I've heard that mentioned a few times. High calcium could be another reason, but mine tested in range. There is also a relation to bile acids.

Anyone else has this tested before?

6 hours ago, Fchawk said:

Hey everyone, this is just a 5 months update from this post. So far, I think that this is method of tackling the side effects of accutane is very effective. It isn't instant, but neither is it expensive or risky in the slightest. Even if you had no health problems doing these things would only improve your health, and most, if not all, of these things you could buy at the supplement aisle of your supermarket, maybe for creatine you may have to go to a supplement store, but I have seen them in supermarkets too, usually just a bit dearer.

Currently I feel I am pretty much 100%, and maybe I am lucky, but I just wanted to let you know it has worked for me, and the post below explains why it should work in greater detail

All in all, I think that I am having a lot of success with this, and encourage any interested to try it as well. Pretty much all the necessary research is up above, and I think that this is the best way to make a full recovery. Good Luck!

Pls be more specific, you're taking all of these products mentiond, or just some of them??

Article talks a lot about brain damage, proven brain damage in fact through testing!! Is that what you've done? All this testing?

This couldhave some significance. This is the acne bacteria. They are more recently looking at drug targeting menaquinones to kill bacteria. This (MQ-9) is a long chain menaquinones similar in structure to mk-7 (K2)
There are more menaquinones in the body then is necessary for k2 production. There might be a purpose for this. mk4 is not significant enough in the diet to contribute to k2 status, meaning there is another process.
They can collect in the skin.
menaquinones, their metabolites and analogs gather and are transported through the lymphatic system. This could be part of innate immunity. The lymphatic system is what fights infection. If this becomes suppressed this could possibly lead to autoimmunity.
Lets call it something similar to a b12 deficiency for example. you have basically months to figure this out before irreversible damage might set in. So imagine something gone or significantly suppressed that was suppose to come back (just like good bacteria after antibiotic treatment) That didnt for various reasons in certain susceptible individuals . This could be why some people spontaneously recover, while others dont.
Im not sure about all this yet, but there is a chance. If so the research and testing isnt up to date on this yet.

https://link.springer.com/chapter/10.1007%2F978-94-017-2803-4_2

The genusPropionibacterium

Abstract

The propionic acid bacteria constitute the genusPropionibacterium, which, together withEubacterium, comprises the family Propionibacteriaceae. The namePropionibacteriumwas suggested by Orla-Jensen (1909), because these bacteria produce large amounts of propionic acid during fermentation. Overall, the propionibacteria are characterized as Gram-positive, non-sporeforming, non-motile, facultatively anaerobic or aerotolerant, rodlike bacteria. They contain menaquinones, mainly MQ-9(H₄) (Fernandez and Collins, 1987), and C₁₅-saturated fatty acids in their membrane lipids. The G+C content in their DNA is in the range of 5367 mol%.

10 minutes ago, guitarman01 said:

This couldhave some significance. This is the acne bacteria. They are more recently looking at drug targeting menaquinones to kill bacteria. This (MQ-9) is a long chain menaquinones similar in structure to mk-7 (K2)
There are more menaquinones in the body then is necessary for k2 production. There might be a purpose for this. mk4 is not significant enough in the diet to contribute to k2 status, meaning there is another process.
They can collect in the skin.
menaquinones, their metabolites and analogs gather and are transported through the lymphatic system. This could be part of innate immunity. The lymphatic system is what fights infection. If this becomes suppressed this could possibly lead to autoimmunity.
Lets call it something similar to a b12 deficiency for example. you have basically months to figure this out before irreversible damage might set in. So imagine something gone or significantly suppressed that was suppose to come back (just like good bacteria after antibiotic treatment) That didnt for various reasons in certain susceptible individuals . This could be why some people spontaneously recover, while others dont.
Im not sure about all this yet, but there is a chance. If so the research and testing isnt up to date on this yet.

https://link.springer.com/chapter/10.1007%2F978-94-017-2803-4_2

The genusPropionibacterium

Abstract

The propionic acid bacteria constitute the genusPropionibacterium, which, together withEubacterium, comprises the family Propionibacteriaceae. The namePropionibacteriumwas suggested by Orla-Jensen (1909), because these bacteria produce large amounts of propionic acid during fermentation. Overall, the propionibacteria are characterized as Gram-positive, non-sporeforming, non-motile, facultatively anaerobic or aerotolerant, rodlike bacteria. They contain menaquinones, mainly MQ-9(H₄) (Fernandez and Collins, 1987), and C₁₅-saturated fatty acids in their membrane lipids. The G+C content in their DNA is in the range of 5367 mol%.

Hey I was actually just about to message you. Do you have any more information on Babis by any chance? I found some posts by him on a dysautonomia site for AAG and his last status talks about how IVig has started to lose its' effect. Yet he won't try immunosuppressive drugs ? Why? I have no idea...

So just wanted to know if you were at all in touch with him.