Acne.org Products
4 hours ago, ACCUiTy_drANE said:The association between depression and hippocampal shrinkage has been known for many years now. Fortunately, this isn't very unique to Accutane:"Such atrophy is centered in a brain region called the hippocampus. This structure plays a critical role in learning and memory, and the magnitude of the hippocampal volume loss (nearly 20% in some reports; refs. 24) helps explain some well-documented cognitive deficits that accompany major depression. . . . Moreover, more prolonged depressions were associated with more severe atrophy." - Source
If that is truly the mechanism in which Accutane causes cognitive problems and depression, it should be fairly treatable. Long-term administration of SSRIs is known to grow the hippocampus. In fact, when that ("brain growing") mechanism is blocked, the therapeutic effects of SSRIs are blocked as well. SSRIs obviously come with their own risks. NSI-189 is another option, but studies conducted on humans show that is grows the amygdala (part of brain associated with fear) to a greater extent than it grows the hippocampus, which may explain the panic attacks. Nevertheless, I found NSI-189 extremely helpful for recapturing emotional depth.
I was reading through government documents and FDA memos and found some fascinating information regarding vitamin A's effect on the brain. For those of us with anhedonia and depersonalization, I believe something far more sinister is going on:
That image sums up three troubling things: 1) Symptoms of acute vitamin A overdose mimic symptoms of schizophrenia, 2) Certain genes implicated in schizophrenia are involved in the retinoid signaling system. The retinoid receptor RXR is involved in the expression of dopamine neurons in the midregion of the brain, a part that is dysfunctional in schizophrenia patients. 3) Retinoic acid binds to the retinoid receptors RAR and RXR, which then eventually regulate the expression of genes pertaining to serotonin and dopamine.
It can be easy to dismiss that research because many of us are not experiencing hallucinations, but many of us have the negative symptoms of schizophrenia. We are unmotivated, feel disconnected from our surroundings, have cognitive dysfunctions, experience anhedonia, and have heavy brain fog. I believe it's possible Accutane uniquely messed up our retinoid receptors (at least for some of us). Although we may not have excess vitamin A in our bodies, signaling is still messed up. I know previous posts alluded to RARs and RXRs; hopefully this isn't redundant.
https://www.ncbi.nlm.nih.gov/pubmed/25302294
I dont want to go searching right now but GHK-Cu has the ability to change retinoid acid receptor expression significantly. I couldn't find any info regarding dose or length of treatment for specifically this, as most ghk-cu studies are for different issues and treatment is usually short term.
7 hours ago, ACCUiTy_drANE said:The association between depression and hippocampal shrinkage has been known for many years now. Fortunately, this isn't very unique to Accutane:"Such atrophy is centered in a brain region called the hippocampus. This structure plays a critical role in learning and memory, and the magnitude of the hippocampal volume loss (nearly 20% in some reports; refs. 24) helps explain some well-documented cognitive deficits that accompany major depression. . . . Moreover, more prolonged depressions were associated with more severe atrophy." - Source
If that is truly the mechanism in which Accutane causes cognitive problems and depression, it should be fairly treatable. Long-term administration of SSRIs is known to grow the hippocampus. In fact, when that ("brain growing") mechanism is blocked, the therapeutic effects of SSRIs are blocked as well. SSRIs obviously come with their own risks. NSI-189 is another option, but studies conducted on humans show that is grows the amygdala (part of brain associated with fear) to a greater extent than it grows the hippocampus, which may explain the panic attacks. Nevertheless, I found NSI-189 extremely helpful for recapturing emotional depth.
I was reading through government documents and FDA memos and found some fascinating information regarding vitamin A's effect on the brain. For those of us with anhedonia and depersonalization, I believe something far more sinister is going on:
That image sums up three troubling things: 1) Symptoms of acute vitamin A overdose mimic symptoms of schizophrenia, 2) Certain genes implicated in schizophrenia are involved in the retinoid signaling system. The retinoid receptor RXR is involved in the expression of dopamine neurons in the midregion of the brain, a part that is dysfunctional in schizophrenia patients. 3) Retinoic acid binds to the retinoid receptors RAR and RXR, which then eventually regulate the expression of genes pertaining to serotonin and dopamine.
It can be easy to dismiss that research because many of us are not experiencing hallucinations, but many of us have the negative symptoms of schizophrenia. We are unmotivated, feel disconnected from our surroundings, have cognitive dysfunctions, experience anhedonia, and have heavy brain fog. I believe it's possible Accutane uniquely messed up our retinoid receptors (at least for some of us). Although we may not have excess vitamin A in our bodies, signaling is still messed up. I know previous posts alluded to RARs and RXRs; hopefully this isn't redundant.
Thank you ACCUiTy, it gives me hope that we can repair these glands.....hopefully.
What are your thoughts on taking Methylfolate?? I got some last week but want to know more about peoples experiences with it before i start to take each day!?
Just found this when looking up if esophillic esophagitis could be related to accutane. Looks like this dude got diagnosed with the same thing after a very large dose of tane. This esophillic condition is capable of happening anywhere in the GI tract.
WadeNovember 18, 2010 at 1:38 pmReply
I am currently 23 years old and reside in Canada and was taking accutane ~10years ago at 240mg/day for 6 months. I had the usual side effects while taking the drug, but started having troubles swallowing soon after. The problem persistened and worsened to the point of not being able to swallow most solid foods. 2 years of hospital and specialist visits was finally diagnosed with eosinophilic esophagitis by a scoping and clipping of the esophagus. (Had done barrium swallow, xrays, digital imaging, diets, etc previous to the scoping) They also scoped the stomach and first part of the intestine and found eight Benign Tumors that they removed. All of these symptoms are related to accutane symptoms. I currently have to take some sort of steroid inhaler that I swallow for 30 days at a time and daily ran-pantoprazole to help with inflammation and esophagitis. Is there any lawsuits in Canada or is this done through the states??? Any info please let me know thanks.
- See more at: http://www.aboutlawsuits.com/settlement-accutane-lawsuit-prior-to-trial-10157/#sthash.XmTAmGUu.dpuf
16 minutes ago, guitarman01 said:Just found this when looking up if esophillic esophagitis could be related to accutane. Looks like this dude got diagnosed with the same thing after a very large dose of tane. This esophillic condition is capable of happening anywhere in the GI tract.
WadeNovember 18, 2010 at 1:38 pmReply
I am currently 23 years old and reside in Canada and was taking accutane ~10years ago at 240mg/day for 6 months. I had the usual side effects while taking the drug, but started having troubles swallowing soon after. The problem persistened and worsened to the point of not being able to swallow most solid foods. 2 years of hospital and specialist visits was finally diagnosed with eosinophilic esophagitis by a scoping and clipping of the esophagus. (Had done barrium swallow, xrays, digital imaging, diets, etc previous to the scoping) They also scoped the stomach and first part of the intestine and found eight Benign Tumors that they removed. All of these symptoms are related to accutane symptoms. I currently have to take some sort of steroid inhaler that I swallow for 30 days at a time and daily ran-pantoprazole to help with inflammation and esophagitis. Is there any lawsuits in Canada or is this done through the states??? Any info please let me know thanks.
- See more at: http://www.aboutlawsuits.com/settlement-accutane-lawsuit-prior-to-trial-10157/#sthash.XmTAmGUu.dpuf
Poor bastard. I got diagnosed last year with herniated esophagus hence why I'm on Pariot to stop acid reflux.
My specialist didnt connect it to Accutane, in fact he said I was born with this hernia but I do wonder when I read articles like this one!!
The inflammation alone we all get is enough for me to want to sue Roche' arse. Ruining lives with no responsibility for it, no justice in this life.
All good stuff. We are definitely scratching the surface or at least in the general area of our underlying issues. We need to raise attention or get more help from students in the universities. it's like all of the scientists are employed by big pharma or too much of assholes to help us.
11 hours ago, vdubz137 said:In the article it states that, "Cellular components and functions of the immune system remain at or near normal levels and are less adversely affected when patients are undergoing chemotherapy and/or taking immunosuppressive medications such as steroids."Retinoids are steroids and if you have taken Acccutane then you have essentially been on chemotherapy... I'm not sure what the effects of Chlorella would be after the fact, but if you click the link it seems like Chlorella (from a clean source) can repair damage done to the brain on a cellular level, due to these harsh drugs. It was an interesting article I wanted to share with the group. I only speak for myself but the all natural route could be the answer. That's not to say it isn't without risk but the less big pharma can shove down my throat the better
Thanks for the further explanation. I checked the link and indeed the article also talks about chemotherapy. and yes I agree the question remains, is the positive effect only if you take Chlorella when you undergo chemotherapy or if Chlorelle is able to repair damages after the therapy has ended.
1 hour ago, Roland1968 said:Thanks for the further explanation. I checked the link and indeed the article also talks about chemotherapy. and yes I agree the question remains, is the positive effect only if you take Chlorella when you undergo chemotherapy or if Chlorelle is able to repair damages after the therapy has ended.
Chlorella is in spirulina yeah - been there done that, it's no cure. Still, taking it wouldn't hurt and you may even feel better forit. Chlorophyll, green smoothies with kale etc - all that stuff is good for you, pain in the arse to make when you're busy in the morning though.
Chlorella in larger doses or by itself might be worth a look?
8 hours ago, TrueJustice said:Poor bastard. I got diagnosed last year with herniated esophagus hence why I'm on Pariot to stop acid reflux.My specialist didnt connect it to Accutane, in fact he said I was born with this hernia but I do wonder when I read articles like this one!!
The inflammation alone we all get is enough for me to want to sue Roche' arse. Ruining lives with no responsibility for it, no justice in this life.
Did you mean hiatus hernia or esophagus gernia and did you solve a problem and how?
BTW: I was also diagnosed with hiatus hernia sliding type
9 hours ago, exitrade said:Did you mean hiatus hernia or esophagus gernia and did you solve a problem and how?
BTW: I was also diagnosed with hiatus hernia sliding type
Yep same thing - both ulcerative oesophagus and hiatus hernia are on report, they are the same thing.
Im not doing anything about it in a direct sense. I am controlling my acid reflux which left unchecked burns that region of my throat/chest.
If I wasn't told about hernia I wouldn't even know I have it - no issue eating or swallowing anything. I went to specialist about acid reflux issue.
I take the product Pariot which works 100% for combating reflux.
1 hour ago, TrueJustice said:Yep same thing - both ulcerative oesophagus and hiatus hernia are on report,
That is a pretty serious form of reflux if you actually had ulcers in your esophagus. These gi issues including chronic reflux are actually some of the symptoms they are lumping together in some of the lawsuits. It's basically ibs going in the other direction. Now that accutane has been off the market for awhile I think the statue of limitations will be running out soon, alot of us are years past this anyways. I wonder if there would be any exceptions if we really found something. Basically like a drug induced skin change from the inside induced by accutane that could be causing some of these symptoms.
54 minutes ago, guitarman01 said:That is a pretty serious form of reflux if you actually had ulcers in your esophagus. These gi issues including chronic reflux are actually some of the symptoms they are lumping together in some of the lawsuits. It's basically ibs going in the other direction. Now that accutane has been off the market for awhile I think the statue of limitations will be running out soon, alot of us are years past this anyways. I wonder if there would be any exceptions if we really found something. Basically like a drug induced skin change from the inside induced by accutane that could be causing some of these symptoms.
Well like I said my specialist reckons I was born with it this hernia, even though I told him about my taking Roaccutane and that's why I have fatigue and gut issues. Either he doesn't believe tane to be the cause or he wants to avoid stating that's what caused it.
I just don't have the energy to sue Roche or pursue any legal avenues, I'm all about trying to fix my health.
If more tane victims happen to have this hiatus hernia - that won't be just a coincidence it'll be something to connect back to when we were on tane.
I wonder if that Canadian guy successfully sued or not??
His case sounded extreme but then how would he prove it resulted from Roaccutane??
It'd be like trying to sue for depression - how do you prove it??
no I dont really believe accutane could cause a physical change like a hiatus hernia. I meant more like a drug induced change somewhere in the gi tract similar to an allergic reaction that could cause chronic inflammation.Thats what could cause excess eosinophils being produced.I was more so speaking for my case and about that other guy i found with eosinophilic esophagitis post tane. If Accutane was capable of causing this, which i dont know for sure, Maybe something similar could be going on with other people. Idk if this could resolve on its own, need short-term treatment, or become chronic and need lifelong treatment.
As far as changes in the brain that could maybe be detected it prob have to be a sensitive test like a PET scan with before and after shots. I mentioned to a neurologist once about getting a PET scan and he made it sound like my insurance wouldnt cover it or i didnt have cause.
52 minutes ago, TrueJustice said:Well like I said my specialist reckons I was born with it this hernia, even though I told him about my taking Roaccutane and that's why I have fatigue and gut issues. Either he doesn't believe tane to be the cause or he wants to avoid stating that's what caused it
Back to the reflux real quick and /or stomach inflammation for some people. if the inflammation was severe enough this might be capable of causing some nerve pressure or pain along some main nerves( like the spine) That could trigger anything from muscle weakness/spasms to joint pain,to chronic headaches.
Even if people dont feel like they got this going on. You might. I've seen alot of people mention histamine type reactions to supplements or weird mucus production. this could just be your body triggering secretions to fight inflammation somewhere in the body.
Everything you're saying is possible - nothing would surprise me about what tane can do to someone.
I have inflammation issues which I really don't know how to treat other than using fish oil, I've tried curcumin but it did nothing.
As I said, I'm all about trying to improve my health but as time goes on and the medical bills rack-up in relation to what I believe are tane related issues, the more I want compensation - why should I or others be out of pocket from the negligence of a company such as Roche??
Im not the type of person who sues but fuck it, these pricks should pay, I didn't sign up for 20 years of unnecessary pain and anguish.
Someone was saying they felt completely normal on prednisone, well thats the most powerful anti inflammatory you can get.
Looking at something more contained that might work similar thats not systemic could be something like aregime from this study.
and i dont mean just for the esophagus but might have a impact on the body as a whole. its a off the wall approach, but has anti-inflammatory impact on multiple fronts.
Eight patients from a single, adult gastroenterology unit with EoE were identified. Diagnosis was made by histological findings in addition to clinical symptoms. All the patients received 12-week treatment with combined oral pantoprazole 40 mg, montelukast 10 mg and swallowed fluticasone 880 micrograms daily. No maintenance medication was routinely used after the treatment.
All patients had eosinophilic infiltration more than 15 per high power field histologically. Five patients (62.5%) had resolved or improved symptoms after the treatment. Two patients (25%) had recurrent but less severe symptoms 2 and 12 months after the treatment, respectively.
I just want to mention that pantoprazole also has anti inflammatory properties. One doctor, as this person mentions has treated many patients successfully with symptoms of IBS with 40mg of pantoprazole daily. I know most of us dont have this type of ibs, but it might be capable still of calming the stomach.
On the flipside of this if accutane was capable of inducinghypochlorhydria, as similar in sjogren's,A ppi is the opposite of what a person would want to do. But if we had reduced stomach acid I think people would be coming up with multiple vitamin deficiencies. but ive had opposing theories on this.
26 minutes ago, TrueJustice said:Im not the type of person who sues but fuck it, these pricks should pay, I didn't sign up for 20 years of unnecessary pain and anguish.
I feel like it be like how they caught capone for tax invasion. Something minor. Like we know what it did. But we dont know what it did. thats the problem. So it have to be something more minor or targeted that was tangible that could be easily proven. like the eye floater thing i was mentioning. like eye floaters caused by reduced lactoferrin in tear tissue caused by accutane. im still going to look into this btw.
1 minute ago, Washer said:I wouldn't touch any anti-acids
unfortunately some people dont have a choice. Truejustice looks like he could be a step away from barrett's esophagus which increases the risk for esophageal cancer.This is definitely something to get under control. but yea accutane is a prime example of risk/reward. just nobody told us we wouldnt see it coming.
7 hours ago, guitarman01 said:I feel like it be like how they caught capone for tax invasion. Something minor. Like we know what it did. But we dont know what it did. thats the problem. So it have to be something more minor or targeted that was tangible that could be easily proven. like the eye floater thing i was mentioning. like eye floaters caused by reduced lactoferrin in tear tissue caused by accutane. im still going to look into this btw. unfortunately some people dont have a choice. Truejustice looks like he could be a step away from barrett's esophagus which increases the risk for esophageal cancer.This is definitely something to get under control. but yea accutane is a prime example of risk/reward. just nobody told us we wouldnt see it coming.
One of the first things I went to doctor about was eye floater issue, that was about 17 years ago. Got dismissed instantly by the doctor at the time when I tried to link it to tane - he even said his brother has eye floaters and got them at a young age, he just said some people get them,some don't.
In relation to my esophagus, specialist doesn't want to see me again as I've responded to Pariet in good way - no more reflux.
I cert hope it doesn't develop into something more sinister!!
In regard to the whole systemic dryness thing many of us have post tane which I'd imagine includes the joint pain and eye floaters etc, I've yet to investigate
Sjogren's syndrome which I know others on here have looked into.
i think we need to look into this more, look at how it's managed and treated.
I think I'm at the point where I now know that no supplement is going to get me out of this mess, it's going to take another drug or multiple drugs to get us out of this hole - prob with that is that they all come with their own list of side effects. Pariet for example can lower ones magnesium levels - thus far my levels are ok thankfully.
In regard to gut, for me probiotics don't do a thing, other than the Pariet helping stop reflux the best and most noticeable product was a "digestive aid" I definitely felt better after eating taking one of these!!
2 hours ago, guitarman01 said:Someone was saying they felt completely normal on prednisone, well thats the most powerful anti inflammatory you can get.
Looking at something more contained that might work similar thats not systemic could be something like aregime from this study.
and i dont mean just for the esophagus but might have a impact on the body as a whole. its a off the wall approach, but has anti-inflammatory impact on multiple fronts.Eight patients from a single, adult gastroenterology unit with EoE were identified. Diagnosis was made by histological findings in addition to clinical symptoms. All the patients received 12-week treatment with combined oral pantoprazole 40 mg, montelukast 10 mg and swallowed fluticasone 880 micrograms daily. No maintenance medication was routinely used after the treatment.
All patients had eosinophilic infiltration more than 15 per high power field histologically. Five patients (62.5%) had resolved or improved symptoms after the treatment. Two patients (25%) had recurrent but less severe symptoms 2 and 12 months after the treatment, respectively.
I just want to mention that pantoprazole also has anti inflammatory properties. One doctor, as this person mentions has treated many patients successfully with symptoms of IBS with 40mg of pantoprazole daily. I know most of us dont have this type of ibs, but it might be capable still of calming the stomach.
On the flipside of this if accutane was capable of inducinghypochlorhydria, as similar in sjogren's,A ppi is the opposite of what a person would want to do. But if we had reduced stomach acid I think people would be coming up with multiple vitamin deficiencies. but ive had opposing theories on this.
My IBS CuredI use to have what was diagnosed as IBS. I would go to the bathroom with loose stool or diarrhea at least 3 to 4 times a day. No matter what I changed in my diet it was there all the time. I hated leaving my house and I never ate when away from the house. I eventually got tired of living this way so I decided to talk to a new doctor and he changed my life. He said that he had given out Protonix to 100's of patients that had IBS and it seemed to work wonders. I was willing to try anything so I said count me in. Within a week I was getting better and after a month I was using the bathroom no more than 1 time a day (normal stool). I know that it is the Protonix because I was forced to change because my insurance wanted me to switch to Prilosac because it wascheaper, when I switched I was back to the loose stool and 3-4 times a day. I went right back on Protonix. It has been 5 yrs now and I am on a bathroom schedule of 1 a day 🙂 I posted this hoping that it might help someone else as I know IBS can really suck the life out of you.....
I'm the one with the prednisone experience. I do have this chronic cough but no actual bile comes up. It's a dry type of cough but happens daily many times. I do also sometimes have days where my throat feels sorta swollen and I get some issues with swallowing but I don't actually get any acid reflux symptoms. Prednisone did make me feel great/almost godly but usually people do have a greater sense of well being on it. This wholeeosinophilic esophagitis thing has me sort of concerned...
My top recommendations for anyone looking to overcome the long-term effects of Accutane:
Read these books by Andreas Moritz and follow the advice as closely as possible (It's timeless Ayurvedic knowledge).
1) Timeless Secrets of Health and Rejuvenation
2) The Amazing Liver and Gallbladder Flush (I recommend doing AT LEAST 15 of them to get all drug residuals out of the liver).
Cleanse the intestines from top to bottom thoroughly with magnesium oxides (like OxyPowder, Colosan, etc.)
Herbal intestinal cleanse products also work but magnesium oxide is more effective and less harmful.
Read Edgar Cayce's Guide to Colon Care book.
Do at least 3 colonics or colemas to cleanse the buildup out of the lower bowel (you must massagethe intestines while holding in the water to loosen it up. For some, it takes 4-5 sessions to start releasing the buildup.
If you do the above, you will regain MUCH of your lost vitality.
If you are still unwell, there are pathogens or subconscious emotional traumas holding you back.
In that case, research chlorine dioxide (MMS) heavily and pure gum spirits of turpentine.
For emotional traumas, either water fasting or Emotional Freedom Technique.
There are many other ways of releasing these, though.
*Also, visit my profile where you may find a few useful posts and comments.
Blessings to all.
Yea, all that good stuff helps anyone with digestive ailments post tane, but the digestive system isn't the cause of the majority of our symptoms. I've been on a vegan diet, done a liver cleanse, and drink kefir every day...helps big time...doesn't cure.
No, the evidence is starting to point towards the brain, receptors, and ultimately the cells.
Some research on one of many of my visual side effects that I deal with every day:
https://en.wikipedia.org/wiki/Visual_snow
Visual snow is now regarded as a unique syndromeusually presenting with other symptoms, such as persistentafterimages,photophobia, enhanced blue field entoptic phenomenonandtinnitus.
Recent research has confirmed a brain hypermetabolism in persons with visual snow, located principally in the rightlingual gyrus.
In May 2014 the results from the first major research trial into visual snow were reported.[4]The study described strong evidence frompositron emission tomographyscans that the disorder is associated with hypermetabolism in the rightlingual gyrusand left cerebellar anterior lobe of the brain. The researchers stated that pinpointing visual snow (and its related symptoms such as afterimages) to a functional problem in a specific brain area may open up possibilities for targeted treatment and that treatment trials will follow.
Isotretinoin Induced Night Blindness
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4533586/
Isotretinoin - It is the only oral drug acting on all the four pathophysiologic factors of acne vulgaris: Androgen-derived overproduction of sebum (seborrhea), ductal hypercornification (closed and open comedones), changes in microbial flora and immunologic processes and inflammation.
^^ (there's some evidence for the Gut theorists. Yes, I look at all the data and discern.)
Isotretinoin was discontinued and the patient was started on vitamin A supplements (25,000 units) daily for 4 weeks. The patient responded completely to vitamin A supplementation (25,000 units) and his night vision recovered completely. Isotretinoin was discontinued and the patient was started on vitamin A supplements (25,000 units) daily for 4 weeks. The patient responded completely to vitamin A supplementation (25,000 units) and his night vision recovered completely.
Our patient developed visual symptoms within 7 days of isotretinoin therapy which resolved after 1 month of vitamin A supplementation. Halpagiet al.reported similar clinical findings in a 21-year-old female student who developed night blindness after 2 weeks of isotretinoin therapy.[5] It has been hypothesized that prior hypovitaminosis A can be a predisposing factor for isotretinoin-induced nyctalopia.[6] Does administration of isotretinoin in patients having low serum vitamin A levels makes them more susceptible to visual problems is a question yet to be answered with robust evidence.
It can be postulated that isotretinoin may act as an antagonist and interfere with vitamin A in the visual pathway.
My sentiments exactly!!
You could do 40 Moritz liver cleanses - you might have a clean liver at the end but you won't be cured from tane related issues.....happy for someone to prove it otherwise!!
Liver cleanse doesn't = repaired brain receptors or normal functioning of sebaceous glands. Again, happy to be proven wrong!!
im going to be looking into this. il get back to you guys when i get more information such as cost. looks like insurance might even cover it.
These eye floaters are annoying as hell some days. BTW I didnt get eye floaters until I started experimenting with vitamin A for the second time. So a word of caution. Also now that I think about it, Accutane had zero effect on my eyes while I was on the drug for 4 to 6 months. It wasnt until quite a few years after accutane I noticed my eyes becoming sensitive to light(forced to wear sunglasses when i never had to before) and decreased night vision.
And I dont just mean Utah for this treatment. Im sure it will become available at more laser eye surgery specialist.
New Laser Procedure Available To Treat Eye Floaters
Issue 3.16
A new laser procedure to treat eye floaters is now available in southern Utah. The procedure is being performed by the Dixie Ophthalmic Specialists at Zion Eye Institute.
Jason Ahee, MD is the Executive Medical Director for Zion Eye Institute. Eye floaters are a very common problem. They usually appear as obstructions in a persons vision. They typically move around but can also be stationary. While not always a serious concern, they can range from distracting and annoying to visually disabling. Our new laser procedure will be a great help for patients struggling with eye floaters, said Dr. Ahee.
Floaters are found in the vitreous body, the clear gel that occupies most of the inside of the eye. They are caused by normal aging changes in the vitreous gel and there is no way to prevent them. Prior to this new laser procedure, the only way to remove floaters was to undergo invasive eye surgery to remove the vitreous gel and the floaters along with it. Most patient were told to just live with it because of the risks associated with surgery.
Laser floater removal, however, can be done with minimal risk. The procedure is quick and painless. It is performed in the office with virtually no recovery time, and it is covered by medical insurances, including Medicare.
The vitreous gel is 99% water and 1% solid elements. Of the solid portion, there are collagen filaments and hyaluronic acid molecules. The ability of the solid elements to retain water molecules decreases with age, causing liquefaction of the vitreous gel. The solid elements coalesce and form condensations. These condensations may float within the liquid vitreous giving the patient a sensation of floaters in their vision.
The same process that causes floaters may cause flashes of light. When the vitreous pulls on the retina which lines the inside of the eyeball like wallpaper the photoreceptor cells in the retina are mechanically stimulated. The retinal cells are incapable of perceiving pain, pressure, or temperature. The only stimulus that the retina responds to is light. So when the retinal photoreceptors experience mechanical stimulation because of the vitreous pull, they send a signal to the brain in the form of disorganized light, which is perceived by the brain as a flash.
With the accumulation of enough liquid vitreous, the vitreous framework collapses and the vitreous completely separates from the retina. This process is called posterior vitreous detachment which can cause large floaters but is otherwise benign.
If the vitreous pulls the retina enough to cause a retinal tear or hole, this can lead to a retinal detachment which may cause vision loss if not repaired quickly. Any time a patient experiences new onset of flashes and/or floaters, it is recommended that they see their eye doctor for a thorough examination.
Posterior vitreous detachment occurs in less than 10% people under 50 years of age but in more than 60% people who are over 70 years of age. It is more common for people who are nearsighted, who have had an eye injury, have undergone eye surgery, or have had inflammation inside the eye.
22 hours ago, ehohel said:I'm the one with the prednisone experience. I do have this chronic cough but no actual bile comes up. It's a dry type of cough but happens daily many times. I do also sometimes have days where my throat feels sorta swollen and I get some issues with swallowing but I don't actually get any acid reflux symptoms. Prednisone did make me feel great/almost godly but usually people do have a greater sense of well being on it. This wholeeosinophilic esophagitis thing has me sort of concerned...
This sounds like it could be silent reflux. It can be extremely hard to discern. But you might have some type of irritation going on with those symptoms.
2 hours ago, guitarman01 said:im going to be looking into this. il get back to you guys when i get more information such as cost. looks like insurance might even cover it.
These eye floaters are annoying as hell some days. BTW I didnt get eye floaters until I started experimenting with vitamin A for the second time. So a word of caution. Also now that I think about it, Accutane had zero effect on my eyes while I was on the drug for 4 to 6 months. It wasnt until quite a few years after accutane I noticed my eyes becoming sensitive to light(forced to wear sunglasses when i never had to before) and decreased night vision.And I dont just mean Utah for this treatment. Im sure it will become available at more laser eye surgery specialist.
New Laser Procedure Available To Treat Eye Floaters
Issue 3.16
A new laser procedure to treat eye floaters is now available in southern Utah. The procedure is being performed by the Dixie Ophthalmic Specialists at Zion Eye Institute.
Jason Ahee, MD is the Executive Medical Director for Zion Eye Institute. Eye floaters are a very common problem. They usually appear as obstructions in a persons vision. They typically move around but can also be stationary. While not always a serious concern, they can range from distracting and annoying to visually disabling. Our new laser procedure will be a great help for patients struggling with eye floaters, said Dr. Ahee.
Floaters are found in the vitreous body, the clear gel that occupies most of the inside of the eye. They are caused by normal aging changes in the vitreous gel and there is no way to prevent them. Prior to this new laser procedure, the only way to remove floaters was to undergo invasive eye surgery to remove the vitreous gel and the floaters along with it. Most patient were told to just live with it because of the risks associated with surgery.
Laser floater removal, however, can be done with minimal risk. The procedure is quick and painless. It is performed in the office with virtually no recovery time, and it is covered by medical insurances, including Medicare.
The vitreous gel is 99% water and 1% solid elements. Of the solid portion, there are collagen filaments and hyaluronic acid molecules. The ability of the solid elements to retain water molecules decreases with age, causing liquefaction of the vitreous gel. The solid elements coalesce and form condensations. These condensations may float within the liquid vitreous giving the patient a sensation of floaters in their vision.
The same process that causes floaters may cause flashes of light. When the vitreous pulls on the retina which lines the inside of the eyeball like wallpaper the photoreceptor cells in the retina are mechanically stimulated. The retinal cells are incapable of perceiving pain, pressure, or temperature. The only stimulus that the retina responds to is light. So when the retinal photoreceptors experience mechanical stimulation because of the vitreous pull, they send a signal to the brain in the form of disorganized light, which is perceived by the brain as a flash.
With the accumulation of enough liquid vitreous, the vitreous framework collapses and the vitreous completely separates from the retina. This process is called posterior vitreous detachment which can cause large floaters but is otherwise benign.
If the vitreous pulls the retina enough to cause a retinal tear or hole, this can lead to a retinal detachment which may cause vision loss if not repaired quickly. Any time a patient experiences new onset of flashes and/or floaters, it is recommended that they see their eye doctor for a thorough examination.
Posterior vitreous detachment occurs in less than 10% people under 50 years of age but in more than 60% people who are over 70 years of age. It is more common for people who are nearsighted, who have had an eye injury, have undergone eye surgery, or have had inflammation inside the eye.
The last line says it all- inflammation inflammation inflammtion!!!
Seems to be the common theme amongst a whole host of different issues post tane.
Thx for this info on eye floaters, I wasn't exactly sure what the cause was - good overview this one!