- Last Week
im going to dump some information here to save it and close a tab.
Its worth a look.
Gut inflammation that spreads to your sinuses and joints.
You need to understand that this is something that could become a part of you, that evades an immune response.
What do you knock down and what do you promote?
you'd have to replace this with a prophylactic measure because otherwise there could just be a resurgence.
just like experimenting with 5ar inhibitors, there could be microbial involvement.
Saw palmetto for example, contains lauric acid.
Inflammation and gastrointestinal Candida colonization - NCBI - NIH
Gut colonization by Candida albicans aggravates inflammation in the ...
Gut colonization by Candida albicans aggravates inflammation in the gut and extra-gut tissues in mice
We examined whether Candida albicans gut colonization aggravates immune diseases in mice. Chronic and latent C. albicans gut colonization was established by the intragastric inoculation of C. albicans in mice fed as part of a purified diet. Allergic diarrhea was induced by repetitive intragastric administration of ovalbumin in sensitized BALB/c mice. Contact hypersensitivity was evaluated by measuring ear swelling after topical application of 2, 4-dinitrofluorobenzene in NC/Nga mice. Arthritis was induced by intradermal injection of bovine type-II collagen emulsified with complete Freund’s adjuvant in DBA/1J mice. C. albicans gut colonization increased the incidence of allergic diarrhea, which was accompanied by gut hyperpermeability, as well as increased infiltration of inflammatory cells in the colon. Contact hypersensitivity was also exacerbated by C. albicans gut colonization, as demonstrated by increased swelling, myeloperoxidase activity, and proinflammatory cytokines in ear auricles. Furthermore, C. albicans gut colonization promoted limb joint inflammation in collagen-induced arthritis, in an animal model of rheumatoid arthritis. These findings suggest that C. albicans gut colonization in mice aggravates inflammation in allergic and autoimmune diseases, not only in the gut but also in the extra-gut tissues and underscores the necessity of investigating the pathogenic role of C. albicans gut colonization in immune diseases in humans.
also I feel there is definitely something going on with blood flow, I can feel the changes.
I tested positive once for antiphospholipid antibodies.
The skin paleness, followed by returning color, eye sensitivity
Examples how this could be microbial based, something that avoids innate immunity.
Activation of blood clotting factors by microbial proteinaseshttps://academic.oup.com/femsle/article/121/3/327/481469
"these results suggest that peripheral and systemic blood circulation may be impaired by ..."
On the meat diet. To me, this is a type of fasting.
There used to be a guy on here, all he'd eat was scallops.
intermittent fasting or a restricted diet is probably a good idea, incorporated into a type of protocol.
- Last Month
going back to what I have recently posted,
The study used 8 billion cfus of bifido infantis in infants.
mixed in breast milk to feed on milk sugars and colonize and correct dysbiosis.
Thats closer to 2 pills of maximum strength Align that would equal 10 billion.
Up until now there hasnt been a viable supplement for this type of dosage of this isolated bacteria that would be cost effective.
Talk about trying to hit the reset button from birth.
what else is in breast milk that I recently mentioned.
Lauric acid and Capric acid.
Both found in coconut oil and some MCT oils.
Both have antimicrobial effects against pathogenic bacteria and yeast.
An example coming from mothers milk should mean its harmless to beneficial microbes.
This next study is a little hard to believe (and a different strain) but you see the potential.
Therapeutic potential of Bifidobacterium breve strain A1 for preventing cognitive impairment in Alzheimer’s disease
Scientific Reportsvolume 7, Article number: 13510 (2017)
It has previously been shown that the consumption of probiotics may have beneficial effects not only on peripheral tissues but also on the central nervous system and behavior via the microbiota–gut–brain axis, raising the possibility that treatment with probiotics could be an effective therapeutic strategy for managing neurodegenerative disorders.
Gene profiling analysis revealed that the consumption of B. breve A1 suppressed the hippocampal expressions of inflammation and immune-reactive genes that are induced by amyloid-β. Together, these findings suggest that B. breve A1 has therapeutic potential for preventing cognitive impairment in AD.
Increasing evidence suggests that probiotics can not only ameliorate physiological functions such as the epithelial barrier, gut homeostasis, and the immune response but may also alter brain function, conferring beneficial effects on psychiatric and neurological diseases13,16. The underlying mechanism, however, remains unclear. Here we showed that oral administration of B. breve A1 prevented cognitive decline in AD model mice, with a reduction in the immune response and neuronal inflammation.
Until now, no clinically successful therapeutic treatment or drug for AD has been reported to date.
These strains need to be looked at on an individual basis.
Even the experts say its trial and error because they just dont know enough yet.
speaking of supplements, I noticed something interesting at the drugstore the other day.
The Align brand probiotic, which is probably one of the highest quality probiotics on the market,
came out with a new formula that's 5x the amount of bifidobacterium infantis 35624 for relatively the same price.
anyone that's looked into align knows how expensive it can be.
maybe they were trying to model this study and dosage, which is 5 billion for infants.
The presence of B. infantis in the intestines of infants is associated with health benefits and these bacteria are nourished by breast milk. However, these beneficial bacteria are present at significantly lower levels in breastfed infants in developed countries than in developing countries.
Mothers and infants in the study received either a B. infantispreparation and lactation support, or lactation support alone, from seven to 21 days after birth. The bacteria quickly established themselves in the babies, crowding out other gut bacteria that are associated with intestinal problems and immune-related diseases such as asthma, allergy and autoimmune disease. Furthermore, supplementation with B. infantis also changed the biochemical composition of infant feces. These beneficial changes lasted for up to a year in babies that were primarily breastfed.
These results suggest a possible method to improve gut microbiome health and prevent immune-related diseases in breastfed infants in developed countries.
then you can look at competition of carbs. bacteria vs yeast.
Feb 16, 2011 - Bifidobacteria can utilize a diverse range of dietary carbohydrates that ..... B. longum subsp. longum BB536 and B. longum subsp. infantis M-63) ...
The influence of dietary carbohydrates on in vitroadherence of four Candida species to human buccal epithelial cells
what else. some more thoughts real quick.
on b vitamins
specifically b3.The current study shows that a C. albicans enzyme, known as Hst3, is essential to the growth and survival of the yeast. Researchers found that genetic or pharmacological inhibition of Hst3 with nicotinamide, a form of vitamin B3, strongly reduced C. albicans virulence in a mouse model. Both normal and drug-resistant strains of C. albicans were susceptible to nicotinamide. In addition, nicotinamide prevented the growth of other pathogenic Candida species and Aspergillus fumigatus (another human pathogen), thus demonstrating the broad antifungal properties of nicotinamide
Theres another antibody.
saccharomyces cerevisiae antibodies
Antibodies to the baker's yeast Saccharomyces cerevisiae (ASCA), recently proposed as a serological marker of Crohn's disease, have also been detected in other autoimmune disorders.
10 hours ago, TrueJustice said:
Sorry but what are you suggesting to do about it??
what’s the follow up plan?
90 bucks through walkin labs.
Fungi and Intestinal Inflammation Finding May Open Door to New IBD Therapies
Weill Cornell Medicine researchers have found that immune cells that process food and bacterial antigens in the intestines control the intestinal population of fungi. Defects in the fungus-fighting abilities of these cells may contribute to some cases of Crohn's disease and other forms of inflammatory bowel disease (IBD), note the scientists.
CX3CR1+ Mononuclear Phagocytes Control Immunity to Intestinal Fungi
Intestinal fungi are an important component of the microbiota, and recent studies have unveiled their potential in modulating host immune homeostasis and inflammatory disease
"After discovering that fungi might be involved in the pathology of IBD, one of the big questions in the field has been how to identify patients who would benefit from antifungal co-therapy, and our finding suggests a way to do that,"
speaking of diet, the most extreme would be elemental.
what you might be doing is starving out microorganisms that are telling you how to think, what to eat,
what you crave, obsessive type behaviours.
you see how microorganisms can control the host if allowed to over populate.
this is fungus.https://www.ncbi.nlm.nih.gov/pubmed/17431821
Coprinus comatus and Ganoderma lucidum interfere with androgen receptor function in LNCaP prostate cancer cells.
For the first time, to the best of our knowledge, we demonstrated that C. comatus and G. lucidum decreased androgen and glucocorticoide receptors transcriptional activity in breast cancer MDA-kb2 cells in a dose-dependent manner, and suppressed androgen receptor (AR) protein level in LNCaP and MDA-kb2 cells. Our findings suggest that AR and non-AR mediated mechanisms underlie the effects of C. comatus and G. lucidum.
A 27-year-old inmate in Hutchinson Correctional Facility, Kansas, died of a fungal infection months after he began complaining about the pain to the prison ...Missing:
self| Must include: selfOct 24, 2011 - When last we met the Wolbachia bacteria, they were being referred to as a "gonad-chomping parasite." That would seem to have negative ...
I dont need to say any more or look into this anymore.
now you test it. Because it raises the question.
Discontinued: Candida albicans Serology and Antigen Tests
As of July 31, 2018, send out testing to Mayo Medical Laboratories for “Candida albicansAntibodies (IgG, IgA, IgM)” and “Candida Antigen Detection” will be discontinued.
Mayo Clinic discontinued these tests due to a lack of clinical significance and challenges with interpretation as compared to fungal culture. Candida antibodies are detected in 20-30% of healthy individuals while immunocompromised individuals at increased risk for systemic candidiasis have blunted antibody responses. Sensitivity of 62% and specificity of 53% have been reported when using antibody levels to diagnose candidiasis.
I tested positive for all the antibodies above.
20 to 30 percent in healthy individuals.
so 70 to 80 percent got something going on.
couple more things.
plenty of people have come down with advanced gingivitis and dental caries/erosion post tane, post fin.
Im one of them.
could be a cause to look into more forms of dental care if true.
Eukaryotic resistance to fluoride toxicity mediated by a widespread family of fluoride export proteins.https://www.researchgate.net/publication/258203436_Eukaryotic_resistance_to_fluoride_toxicity_mediated_by_a_widespread_family_of_fluoride_export_proteins
Although ﬂuoride is plentiful in the environment and is commonly used at high concentrations in oral hygiene products,little has been known about how biological systems overcome the toxic effects of this anion. We demonstrate that a protein called FEX in many fungi is essential for cell survival in the presence of high ﬂuoride concentrations. The protein is required for the rapid expulsion of cytoplasmic ﬂuoride, indicating that many eukaryotic species that carry FEX genes likely avoid ﬂuoride toxicity by purging cellular ﬂuoride.
Does Oral Isotretinoin Prevent Propionibacterium acnes Resistance?https://www.karger.com/Article/Pdf/246012
This paper reports preliminary data demonstrating that oral isotretinoin (Roaccutane®/Accutane®) significantly reduces total numbers of resistant P. acnes on the skin of all patients.
So what about other bacterial resistance? possible?
"These compounds exhibited significant antibacterial activities against the Gram-positive bacteria Staphylococcus aureus, Bacillus subtilis, S. epidermidis, and Propionibacterium acnes. They also exhibited antiandrogen activities."
Gut Microbiota and Metabolic Health: The Potential Beneficial Effects of a Medium Chain Triglyceride DietMay 12, 2016 - Here, we describe how dietary medium chain triglycerides (MCT), .... Gut microbiota is now considered as a key factor of metabolic health [44 ...
"we believe that gut microbiota structure may not only serve as a biomarker of those metabolic states, but can also be subjected to a diet-induced remodelling, modifying in turn the metabolic status of patients (MUHO vs. MHO or lean state). Dietary MCT, taken alone or with other supplements (such as prebiotics, probiotics, organic acids, etc.) could be used as anti-obesity interventions, in regards to their capacity to prevent intestinal permeability/endotoxemia by remodeling gut microbiota, and to prevent unhealthy storage by improving the lipid catabolism/anabolism balance"
The 5ar inhibitor is supposedly the lauric acid in coconut oil and the free fat (rancid) at that.
This is mostly removed from most mct oils.
mct oil only contains capric and caprylic acid.
you can also find a study on coconut oil boosting testosterone and testicular weight.
Everything conflicts as always.
Trial and error (caution) as always.
sounds about right, or at least the from hell part.
I bought organic virgin coconut oil for grilling my steaks. to my surprise, after a few days I started to notice an improvement in digestion. continued to use for weeks and eating raw 1 tablespoon. suddenly, I began to notice an increase in skin oils, hair loss, increase in libido, more rigid erections.
do not know why. I have some hypotheses:
(1) 5alpha reductase inhibition
coconut oil is DHT inhibitor. Exists a rebound effect in the production of enzymes 5alpha reductase after inhibition. after consuming the body produces more enzymes .
(2) Autoimune relief
The auoimune system inhibits the 5alpha reductase and coconut oil is a 5alpha reductase. The autoimune system stop to inhibits the enzime.
(3) boost thyroid. I remember when I used T4, increased skin oiliness, but remember not to increase libido and erection.
(4) Kill Candida
PFS is the source of an infestation by candida.
started to recover well in time that I started using coconut oil.
The Dietary Food Components Capric Acid and Caprylic Acid Inhibit Virulence Factors in Candida albicans Through Multitargeting.
Vitamin Biosynthesis as an Antifungal Target - NCBI - NIHby Z Meir - 2018Jump to The Vitamin A, C, D, E, and K Pathways Are Not Suitable as Antifungal ... - They include the vitamin A, C, D, E, and K pathways.
This is kind of interesting.
Objective: The microbial transformation of finasteride by fungus has been investigated to obtain biologically more potent derivatives.
Im going to keep on moving here.
Some science mixed with thought.
Ive seen enough recent studies to see this is emerging as a very real prospect.
Not just some natural healing type pseudoscience.
You have to imagine how hard this could be to treat as well if allowed to take hold.
Take h. pylori for example. A stomach bacteria that requires triple antibiotic therapy to knock it out.
And yeast is more sophisticated than that. (that meaning bacteria)
PATHOGENESIS OF CANDIDA ALBICANS IN ANDROPAUSAL HORMONAL IMBALANCE
- More Than a Month
This is what i've been looking at.
Yeast worsens gut disorder
Yeast in the gut boosts levels of uric acid, which damages the intestinal wall and worsens bowel inflammation in mice.
Gut bacteria are known to influence intestinal disease, but the role of gut fungi — such as the yeast Saccharomyces cerevisiae (pictured) has not been well studied. June Round and her colleagues at the University of Utah in Salt Lake City fed mice with S. cerevisiae, then chemically induced gut inflammation, or colitis. Mice that were given the yeast produced more uric acid and had more gut inflammation than untreated animals did. The team found that the yeast increased the intestinal wall's degradation of molecules called purines, leading to higher uric acid levels. Treating the animals with allopurinol, an inhibitor of purine metabolism, eased the colitis.
In serum samples from healthy humans, elevated uric acid levels correlated with greater numbers of antibodies against S. cerevisiae. This yeast might play a part in human inflammatory bowel disease, the authors suggest.
Probiotic normalization of Candida albicans in schizophrenia: A randomized, placebo-controlled, longitudinal pilot study.
The molecules and pathways of the gut-brain axis represent new targets for developing methods to diagnose and treat psychiatric disorders. Manipulation of the gut microbiome with probiotics may be a therapeutic strategy with the potential to relieve gastrointestinal (GI) comorbidities and improve psychiatric symptoms. Candida albicans and Saccharomyces cerevisiae, commensal yeast species, can be imbalanced in the unhealthy human microbiome, and these fungal exposures were previously found elevated in schizophrenia.
...In conclusion, the administration of probiotics may help normalize C. albicans antibody levels and C. albicans-associated gut discomfort in many male individuals. Studies with larger sample sizes are warranted to address the role of probiotics in correcting C. albicans-associated psychiatric symptoms.
Opportunistic pathogen Candida albicans elicits a temporal response in primary human mast cells
Yeast Infection Linked to Mental Illness - 05/04/2016May 4, 2016 - In a study prompted in part by suggestions from people with mental illness, Johns Hopkins researchers found that a history of Candida yeast ...
Gut dysbiosis promotes M2 macrophage polarization and allergic airway inflammation via fungi-induced PGE₂.
Manipulation of Host Diet To Reduce Gastrointestinal Colonization by the Opportunistic Pathogen Candida albicans
Study in Mice Suggests Coconut Oil Can Control Overgrowth of a Fungal Pathogen in GI Tract
On 7/9/2016 at 4:46 PM, eph95 said:
Every few months I come back to this thread to take a look and see how people are doing. And it's quite sad to see, but the majority of people are wasting their time and going the wrong way about it. If you want to cure your Isotretinoin side effects, it's actually quite simple. I see so many people, talking about different theories on how Isotretinoin causes negative side effects and health issues after ceasing the drug. I am quite shocked to see people are actually still talking about DHT levels and how this is one of the mechanisms by which Isotretinoin damages the body. There is also the copper theory, amongst many others. But everyone is wrong! People seem to over complicate things.
if you really want to cure your depression, suicidal thoughts, mood swings, erectile dysfunction, low libido, fatigue and all the rest then please. Don't listen to most people on this thread.
the key to repairing the chemotherapy damage from Isotretinoin. Is by addressing your gut. The way accutane causes all these health problems is because it is a very powerful agent, it wipes out all the bacteria in your gut (that's the good bacteria too). You need to replenish your healthy gut flora. There is a Reason why the gut is called "the second brain", it's because it produces 90% of the bodies serotonin. Isotretinoin also weakens the gut lining and causes crippling candidiasis. The only way to cure yourself is to take a combination of natural antifungals and probiotics . If anyone wants any more info then email me firstname.lastname@example.org
have a good day everyone and don't give up.
ed, Oxford, United Kingdom.
Not to shift gears because submitting 23andme data is a good idea or no brainer,
but keep this in mind as well.
On 8/13/2018 at 6:56 PM, Dubya_B said:
There's one very easy way to find out if many of us share this polymorphism. If you do have 23andMe data, please upload it and take part in the propeciahelp GWAS.
Yes. Easy enough to do if you already have the kit. if not 100 bucks for 23andme is a good investment.
Speaking of 100 bucks,
it would be nice to see someone else get this test I just posted if you think this could be "product of your environment" based as much as genetics. which i think it could be both.
Meaning what you consumed (Drug), altered your microbial environment.
Shifts that continue to go in the wrong direction.
It would be good to verify this isnt flagging with someone else.
if you have a nearby quest diagnostics, it will cost you 90 dollars through walkinlabs.On 8/10/2018 at 1:27 PM, guitarman01 said:
CANDIDA ALBICANS AB TXC
C.ALBICANS IGG 1.3 H
C.ALBICANS IGA 1.4 H
C.ALBICANS IGM 1.1 H
Along with 23andme (which i've looked at pretty extensively myself), one thing i'm curious about is if our genes could predict some of our microbiome composition and or vice versa.
Im actually more curious of this atm, which I plan to get the most comprehensive version of this test as soon as I have some more information.
- comprehensive access to your entire microbiome, including viruses and fungi
- Functional metagenomics information, such as insights into what key metabolites and vitamins your bacteria are synthesizing for you
- More comprehensive probiotic bacteria
- Downloadable raw data for every organism detected in your sample
Association between leptin gene rs7799039 polymorphism and lipid profile changes induced by isotretinoin treatment in acne patientshttps://www.dovepress.com/association-between-leptin-gene-rs7799039-polymorphism-and-lipid-profi-peer-reviewed-article-TCRM
Not sure about this yet. But every single antibody was above normal. Not by alot though.
You see it's also found in a percentage of the normal population.
Test Name In Range Out Of Range Reference Range Lab
CANDIDA ALBICANS AB TXC
C.ALBICANS IGG 1.3 H
C.ALBICANS IGA 1.4 H
C.ALBICANS IGM 1.1 H
REFERENCE RANGE: <1.0
<1.0 Antibody not detected
> or = 1.0 Antibody detected
Systemic candidiasis is often characterized by
markedly elevated levels of IgG, IgA, and IgM
recognizing Candida. However, interpretation of
Candida antibody levels is complicated by
detection of antibodies in 20-30% of healthy
individuals, and blunted antibody responses in
immunocompromised patients at risk for systemic
candidiasis. Candida antibody results should be
considered within the context of clinical findings
and results from other relevant laboratory tests,
such as Candida antigen detection and/or culture.
This test was performed using a kit that has not
been cleared or approved by the FDA. The analytical
we will see. maybe there has never been a targeted approach.
im not looking at a all in one "candida" supplement.
As a actual drug I could reference 100mg of fluconazole per day for 3 weeks.
im looking at something similar to sibo, instead with fungus.
Im not looking at candida specifically but its cousins or close relations as well.
Example, saccharomyces cerevisiae. A commensal yeast since birth, can become pathogenic.
im looking at something that is isolated or contained to the lower gi tract but still causes a systemic response.
On 6/25/2018 at 7:23 PM, guitarman01 said:Antifungal properties of selective serotonin reuptake ... - Oxford JournalsIn conclusion, our in vitro studies clearly demonstrate antifungal effects of SSRIs. Animal studies are needed to evaluate the potential role of these psychotropic ...
aac.asm.org/content/58/10/5855.full.pdfMay 16, 2014 - clinical utility of finasteride in the prevention of urinary candidiasis is ... with standardantifungal drugs, and finasteride is one of several.
by E Campione - 2016 - Related articlesApr 29, 2016 - Recent reports hypothesize that the antifungal efficacy of all-trans retinoic acid (ATRA) is mainly related to its strong capacity to stimulate ...
Inflammation. This could constrict blood flow.
Maybe thats why some have found temporary relief taking again what might have harmed them in the first place. But then maybe they end up even worse then they started.
Its been awhile since ive gotten any tests.
But im getting one tonight. Just in case.
im actually looking for a immune response coming from the gi tract.
There's no other way to possibly test this atm.
www.questdiagnostics.com › For Physicians & Hospitals › Test Center
UCLA scientists identify a new way to activate stem cells to make hair growMirabai Vogt-James | August 14, 2017
In this study, Christofk and Lowry, of Eli and Edythe Broad Center of Regenerative Medicine and Stem Cell Research at UCLA, found that hair follicle stem cell metabolism is different from other cells of the skin. Cellular metabolism involves the breakdown of the nutrients needed for cells to divide, make energy and respond to their environment. The process of metabolism uses enzymes that alter these nutrients to produce “metabolites.” As hair follicle stem cells consume the nutrient glucose — a form of sugar — from the bloodstream, they process the glucose to eventually produce a metabolite called pyruvate. The cells then can either send pyruvate to their mitochondria — the part of the cell that creates energy — or can convert pyruvate into another metabolite called lactate.
The research team first blocked the production of lactate genetically in mice and showed that this prevented hair follicle stem cell activation. Conversely, in collaboration with the Rutter lab at University of Utah, they increased lactate production genetically in the mice and this accelerated hair follicle stem cell activation, increasing the hair cycle.
Still a very popular drug as you can see.
Hopefully this will also mean continued research.
For anyone praising the drug, yea I was there once too.
(I just think those posts in this spot might be in the wrong place. We all know what Accutane can do)
Even thought about taking a second course after some zits came back.
Then some things started to happen probably within a year of finishing my course.
The first real sign that something abnormal was going on was probably the total diffuse hair loss that spanned the entire body.
Also the facial flushing, headaches, feelings of warmth in my head( inflammation?) trouble concentrating.
For some the acne is so severe no cream is going to do a thing.
The problem must be fixed internally.
At least even the drug companies might be looking for alternatives.
Isotretinoin Drugs Market Expected To Reach USD 1567.1 Million with Major Players Mylan N.V. (US), Ranbaxy Laboratories Inc, a SUN PHARMA company. (India) and Teva Pharmaceuticals USA by 2023
The Global Isotretinoin Drugs Market has been evaluated as steadily growing market and it is expected that the market will continue to grow similarly in the near future. Increasing prevalence of various skin disorders, genetic disorders and cancer are major driving forces for the market. The market for isotretinoin drugs was around US$ 1257.0 million in 2016 and is expected to reach US$ 1567.1 million which is projected to grow at a CAGR of 3.2% by 2023.
Isotretinoin drugs causes some serious side effects such as birth defects and depression. To avoid these kind of situation, drug manufacturers are focusing more on new drug development. Due to strict regulatory policies, all the top players are trying to improve the efficacy and safety of isotretinoin drug.
On 7/15/2018 at 10:11 AM, Devolution said:On 7/14/2018 at 1:24 PM, guitarman01 said:
This looks like a fairly new method of testing. I wonder how easy this test would be to come by.
btw regarding tests.
you had someone show some low grade brain inflammation going on from a Mayo clinic analyzed PET head scan.
he also tested positive for a pretty rare antibody.
acetylcholine ganglionic neuronal antibody.
So did I.
I tried looking into this a little bit. No local drs are familiar with it. Even including a nero at UW Madison.
This can also affect the gastro system.
it basically could be a autoimmune attack on the nervous system.
Don't quote me on that.
Getting to a place like Mayo is prob a good idea. After you have some type of case built up or maybe some idea of what might be going on.
I might try in the future, I dont have insurance atm.
switching gears been looking at a couple things
a hormone that seems to regulate alot
inhibits digestion?Somatostatin, also known as growth hormone-inhibiting hormone (GHIH) or by several other names, is a peptide hormone that regulates the endocrine system and affects neurotransmission and cell proliferation via interaction with G protein-coupled somatostatin receptors and inhibition of the release of numerous secondary ...
- Somatostatin is homologous with cortistatin (see somatostatin family) and suppresses the release of gastrointestinal hormones
- Decreases the rate of gastric emptying, and reduces smooth muscle contractions and blood flow within the intestine
- Suppresses the release of pancreatic hormones
- Suppresses the exocrine secretory action of the pancreas
on the other side of the spectrum.
Special cells called “Leydig” use lactic acid to suck up cholesterol and convert it into testosterone.
Without lactic acid... your body refuses to do any of the exciting things such as producing more testosterone, growth hormone and energy.
its also a powerful antibiotic thats looking to replace "antibiotics"
you can find it in things like palmolive antibacterial soap.
Although glucose is usually assumed to be the main energy source for living tissues, there are some indications that it is lactate, and not glucose, that is preferentially metabolized by neurons in the brain of several mammalian species
lactic acid is also produced by some commensal bacteria