hello ppl, i will cut to the chase without any introduction,so we have known that scarring is a menifestion of a battle that takes place because our immunity due to the overactivity of fibroblast and the mediators of inflammation,supposing we apply strong antinflammatories on an active lesion/injury,could we arrest the mediators from causing scar,,,,,for eg if not corticosteroids since they are notorious for skin thinning abilities hence atrophy,can we not use something like tacrolimus or mitomycin to hamper fibroblasts and other agents via their strong antinflammatories effects? this should also work as excellent adjuvent to regenerative procedures either?