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ForeverAndPatience

Dissolve Scars With... Bee Stings?!?!

"Bee venom contains hyaluronidase, which attacks scar tissue and dissolves it. In bee venom therapy or sclerolysis, a therapist mixes venom with a saline solution or sterile water. The therapist targets sensitive or painful areas of the scar and makes several injections across the surface of the scar. Even people allergic to bees still benefit from the therapy, but the therapist takes precautions to prevent an allergic reaction. Some therapists place live bees on the body to control the bee stings."

And the article: http://www.livestrong.com/article/11271-reworked-need-dissolving-scar/

Pretty interesting. Has anyone heard of this one?

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There was a point where people thought bee stings cured everything. There was even a King of the Hill episode parodying the phenomenon.

It turned out bee stings don't do jack squat.

It might temporarily help scars by swelling the skin abnormally, thus the scars "disappear" because they're stretched, but this is otherwise pure bupkis.

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There was a point where people thought bee stings cured everything. There was even a King of the Hill episode parodying the phenomenon.

It turned out bee stings don't do jack squat.

It might temporarily help scars by swelling the skin abnormally, thus the scars "disappear" because they're stretched, but this is otherwise pure bupkis.

Dont be so sure dude.

Cool stuff

Melittin attenuates liver injury in thioacetamide-treated mice through modulating inflammation and fibrogenesis.
Source

Department of Pathology, Catholic University of Daegu, College of Medicine, 3056–6 Daemyung 4-Dong, Nam-Gu, Daegu 705–718, Republic of Korea.

Abstract

Liver fibrosis represents a process of healing and scarring in response to chronic liver injury. Following injury, an acute inflammation response takes place resulting in moderate cell necrosis and extracellular matrix damage. Melittin, the major bioactive component in the venom of honey bee Apis mellifera, is a 26-residue amphipathic peptide with well-known cytolytic, antimicrobial and proinflammatory properties. However, the molecular mechanisms responsible for the anti-inflammatory activity of melittin have not been elucidated in liver fibrosis. We investigated whether melittin ameliorates liver inflammation and fibrosis in thioacetamide (TAA)-induced liver fibrosis. Two groups of mice were treated with TAA (200 mg/L, in drinking water), one of the groups of mice was co-treated with melittin (0.1 mg/kg) for 12 weeks while the other was not. Hepatic stellate cells (HSCs) were cultured with tumor necrosis factor α in the absence or presence of melittin. Melittin suppresses the expression of proinflammatory cytokines through the nuclear factor (NF)-κB signaling pathway. Moreover, melittin reduces the activity of HSCs in vitro, and decreases the expression of fibrotic gene responses in TAA-induced liver fibrosis. Taken together, melittin prevents TAA-induced liver fibrosis by inhibiting liver inflammation and fibrosis, the mechanism of which is the interruption of the NF-κB signaling pathway. These results suggest that melittin could be an effective agent for preventing liver fibrosis.

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