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willow569

Member Since 07 Apr 2006
Offline Last Active Aug 09 2011 04:00 PM

#2190325 big pimple with multiple heads?

Posted by willow569 on 12 February 2008 - 08:14 AM

If it doesn't go away soon, you could try getting it extracted by a dermatologist or an esthetician who has experience treating acne.  Is the spot inflamed or red?


#2063844 Nizoral

Posted by willow569 on 15 October 2007 - 10:38 AM

QUOTE(allornothing83 @ Oct 15 2007, 01:33 AM) View Post
I was on minocycline 2 weeks ago, and broke out hideously after a week on my back and neck, shoulders and chest.  From my symptoms it seemed like I had folliculitis.  I read on this site about tyring Nizoral.  Well after 3 days of using it, what I thought was bacne and body acne is almost totally gone!  If you're not sure, you should give this a try.  I have always suffered from back acne but turns out it has been folliculitis, and for now this treatment is working.  It HAS just been 3 days, but I'm hoping this is a long term cure!  Just wanted to share for anyone to take a look at this.


I used it as well and it cleared me up very quickly.   However, after about 3-4 wks it stopped working as well and the folliculitis started coming back (not to say that it will do that for you).  From what I have read about others' experiences, folliculitis tends to come back over and over again.  I have added a couple other treatments to my regimen to deal with the folliculitis - so these might be some things for you to try if the Nizoral stops working.  

First, I still use the Nizoral every few nights.  I also wash with the ZNP bar in the mornings.  Every other night I use a 10% mandelic serum on my chest and back - its antifungal and antibacterial.  I also am on a short term course of oral antifungal medication.  Things seem to be clearing up again - hopefully it will be more long term for me this time.  We shall see....


#2021157 Anything Particular Cause Pustules?

Posted by willow569 on 03 September 2007 - 05:28 PM

QUOTE(ShockBotkins @ Sep 3 2007, 04:14 PM) View Post
It seems all the pimples I get are pustules. I don't get any of the cysts or anything like that, mine usually last for 2-5 days. The thing is I usually have 3-5 on each side of my face around the jawline by my mouth. Is there anything in particular that causes pustules? I've been getting them since I was 13 or 14 and I'm 21 now. Thanks.


Pustules are a form of inflamed acne - some people are more prone to this than others.  Usually, pustules start off as non-inflamed, closed comedones that get infected with bacteria and burst open the follicle wall, which causes an inflammatory response by the body.  Some people only get papules, which are somewhat deeper, inflamed bumps without pus.  A pustule happens with the body pushes the acne impaction to the surface of the skin, along with the pus (which is dead bacteria and white blood cells).

If you are dying for more details, check out this thread:
http://www.acne.org/...ns-t168987.html

There are some things you can do to stop non-inflamed comedones from getting inflamed.  Using a regular exfoliant to keep your pores clear (glycolic or salicylic acid and OTC or prescription retinoids), and something to kill the bacteria (e.g., benzoyl peroxide).   There are other things that can worsen inflamation as well - stress, some dietary factors such as iodine.  Ice and an antiinflamatory pain reliever can also help reduce inflamation.


#2020541 Good overview of different acne lesions

Posted by willow569 on 03 September 2007 - 09:30 AM

I posted this in response to a question in another thread, but I thought this info was worth repeating in its own topic thread.  This is one of the clearest descriptions of the differences between each type of acne lesion and what leads to each type of acne lesion. It also has a nice, concise explanation of why some lesions get inflamed and others don't.  

This is an excerpt from Marc Lees book "Skin Care Beyond the Basics" Great reference book!


Noninflammatory and Inflammatory Acne Lesions

As cells build up on the inside of the follicle wall they form a small impaction called a microcomedo. Microcomedones are actually a mixture of dead cells, bacteria, fatty acids from sebum, and other cellular debris. Microcomedones are not visible to the naked eye. They cannot be seen without a microscope. They continue to retain more and more of the mixture of dead cells, sebum, and bacteria, until they become a visible lesion under the surface of the skin.

There is a point in the development of the impaction where the microcomedo either becomes an inflammatory or noninflammatory lesion. Noninflammatory means that the impaction is not red or inflamed. Examples of noninflammatory lesions are open comedones (blackheads) and closed comedones (whiteheads).

Open comedones occur when the follicle is large enough to hold all of the debris retained by the follicle. The ostium, or opening, in these follicles is dilated by the mass of the impaction, allowing the comedo to push toward the surface opening.

Proprionbacterium acnes (p. acnes) is the scientific name of the bacteria that causes acne vulgaris. Open comedones do not encourage development of this bacterial growth because the follicle opening is large enough to expose the follicle to oxygen. The oxygen is also what causes the blackhead to form at the exposed part of the impaction. This darkening is caused by the exposure of the top of the comedo to the oxygen in the air outside the follicle. The sebum turns a brown color, similar to the way mayonnaise will turn yellow if left out on a picnic table for a period of time. The darkness is also caused by clumps of melanin (skin pigment) present in the dead cells in the comdeo. This theory is easily demonstrated by observing an extracted open comedo. It is a solid cylindrical plug, topped by a dark area that gets lighter as the deeper parts of the impaction are extracted.

Open comedones, therefore, rarely develop into inflammatory lesions. Unfortunately, the same cannot be said for closed comedones. Closed comedones have very small pore openings, which prevents oxygen from readily penetrating the follicle. The walls of the follicle stretch to hold the contents of the impaction, but the follicle opening does not. Because of this lack of oxygen, the lesions can easily become inflamed due to the increasing numbers of bacteria multiplying in the anaerobic environment.

Closed comedones are easily recognizable. They are small underground bumps and are not easily extracted. They are frequently associated with the use of comedogenic cosmetics, as indicated in the blush line of some women.

When enough bacteria form inside the closed comedo and the impaction becomes large enough, a small tear occurs in the follicle wall, which stimulates the immune system to investigate, releasing white blood cells into the area. This begins the inflammatory process.

A papule is a red, sore bump without a whitehead (no pus). This is the beginning of the rescue by the white blood cells. When enough white blood cells arrive, they may form a clump and rise to the surface, creating what is known as a pustule. Pus is the common name for this clump of white blood cells. For practical purposes, a papule is often described by the client as a large, red, sore bump that never comes to a head. Papules seem sometimes to magically disappear. This is because the immune system has won the battle and disposed of the remains through normal blood excretion. Papules affect the nerve endings more than pustules because they are deeper in the skin. This explains the soreness. Pustules have migrated the impaction toward the skin surface, dilating the follicle opening and relieving the pressure on the nerve endings, resulting in less pain.

A nodule is similar to a papule, but is deeper in the skin and feels very solid and sore. Cysts are deep infections caused by a deep, massive invasion of white blood cells. They are very pustular and very large.