wow i actually wanted to ask the same exact question like a day ago! :0what is in our bodies before puberty that allows it wont be inflammed then?why do other ppl dont even get no acne during puberty?the same goes with allergies and intolerances that are popular here, dont u have those from when ur born not appearing some random time at ur life?when i was small could eat dairy, grains etc just fine somehow..only had somekinda alergy from oringes (but not allways either?), would break out everywhere, but those were not acne nd it wasnt just on face (like usually it is first and only place where acne starts)now ill do diff crazy health cleanses and do clean diets, supplemetns etc etc and still break out like its nothing. max few days without new breakouts. but then again i was on bad diet and also will get good skin days. diet improves skin quality a bit i think but it seems so freaking useless for acne..
It's the immune system. At some point, the immune systems of most people who have acne gets messed up. There are lots of ways to accomplish this. And the immune system is so modular that it is conceivable that these problems can just pop up out of seemingly nowhere (though I doubt this is the case with acne).
I think it's useful to catch a glimpse of the broader picture here. Acne starts at puberty for many people - but not for all people. Let us assume that there is something about puberty that allows for acne to form, given the right conditions (a fair assumption I think). For those people who begin to get acne right as they hit puberty, it follows from the model I describe that these people were suffering from pre-existing immunological conditions - an immune response that was always there, but could not manifest itself until the crucial puberty factor came into play.
Now the other people - those who get acne at some later age, be it 14, 15,18, or 25. These people already have the puberty factor. So when these people get acne, the model would predict that the cause is a 'random incidence', by which I mean an immunological change that occurs, as you describe, seemingly randomly. If we consider for a moment that these 'random incidents' are actually randomly happening, the model would predict that most acne sufferers begin to get it around puberty, because puberty would essentially be revealing 12 years of random immunological incident. So you see, it does make sense.
Exactly. People for the most part when they're allergic to something have the allergy rear during childhood - not adolescence. I think it's pretty clear for those of us with good common sense that acne isn't a good allergy.
I could be be wrong but I think a lot if the theories coming from this section of the forums are propelled by pseudo science.
Acne is certainly not an IgE-driven allergy such as a peanut or egg allergy seen in childhood. But there are many other antibodies involved in immunity, and whole sections of the immune system that don't bother to use antibodies. There are even other types of hypersensitivity - types 1 through 4. It is conceivable that acne, which is without question a physical manifestation of an immune response, could in some cases be propagated by food antigens, even using one of our existing definitions of hypersensitivity.