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Acne - Diet Studies (and results)

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I've already read some of the responses regarding Dr. Mann and his affliations but what about the other studies? There's the evidence provided through these studies which members rebuff and of course there's plenty of our own...anecdotal or testimonial evidence over in the Nutrition & Holistic Forum. For example, here's an excerpt from a post by Minnymouse in March 2005:

Here's some excerpts from his emails when I questioned what the study was about:

--We are finding different people have different levels of sensitivity

but your sons diet of moderate level of carb foods and the type (most

sound low GI) should be very good. Look for low GI labelled foods and

get a book that lists GI of foods such as Jennie Brand-Miller's various

GI books, but there are many others. The brown rice is probably the

only thing you mention that could be still high GI (there are even

different types of brown rice) Our subjects ate limited amounts of

whole grain products and limited amounts of rice (low GI basmati) but no

high GI carb foods. I think whole grain low GI foods, should be OK

along with meats and fish (I doubt if the fat at this stage of life is

a worry for your son for acne, weight gain or heart disease, may be

later in life), in fact he may need extra fat and protein to provide him

more energy to make up for the decreased carbs. Certainly avoid sugary

sweet items like sweetened fruit juice and sodas and any product made

from flour or potatoes. Lots of vegetables, fruits and meat and fish

and nuts and beans, and soy. If he seems also sensitive to dairy as

some teenagers are then stay off dairy until acne phase is over.

--We are nearing completion of our study and will publish this year.

Slow process because we not only need to show an improvement in visual

acne condition but blood chemistry changes that can be used to develop

an hypothesis as to what aspects of metabolism are being altered by diet

to effect an acne change. This blood analytical work is happening now

and we are seeing complex metabolic changes that relate to various skin

cell abnormalities and acne. The dermatology side did show improvement

on our diet but I need to qualify this a little. Young boys do not

stick well on diets when they are free living and exposed to abundance

of processed foods. The boys on low glycaemic load diets did gradually

improve (4-8 weeks) if they kept to the diet which included:

Low starch and sugar intake, ie eliminate all high GI foods, which

eliminated most bread, potatoes, most rice, baked goods made from any

flour ie any refined carb product, some fruits also can have high carb

eg bananas so go easy on these, sweetened products with much added sugar,

glucose or corn syrup are very bad even fruit juices with added carb

sweeteners should be avoided.

Our diets were rich in meat, fish, vegetables, salads, most fruits,

nuts and very low in grain and sweetened foods.

The unfortunate part is that it takes several weeks to clear up the acne but only a day or two to reactivate if a high GI carb food is consumed, this we cannot

alter, its just the way the metabolic process works, so the person must

be careful.

Finally we are not all genetically the same and your sons acne may not

respond to the same extent as others and there seem to be some rare

cases where the person has an intolerance to a particular food or food

group that stimulates acne for them individually, dairy is an example

for some people, so if the above does not work try food eliminations

over several weeks and try and identify any food group that he may be

reacting to.


Minny talking:

I sent him an email telling him how much improvement I saw in my son's cystic acne from being on this for 1 month. He has one breakout on his cheek right now verses a whole face full prior to this diet. He is on NO medication.

Here's Dr. Mann's last email- he is reffering to my son's success.

--Very happy to hear this, fantastic for us researchers when we hear the

beneficial results of what we are working on, particularly when it does

not involve drugs.

Once we publish our study we will be talking at conferences in the USA

about it. I also hope to get print and TV media coverage so we can get

this knowledge out to millions of other young people and their families.

If you ever get an opportunity to present this information to media

particularly your US TV shows go for it and let me know. This is really

the best way to to communicate with the widest possible audience and as

you have seen the benefit can be outstanding.

My best wishes for your sons full recovery and his future.

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The sections that I colored within the prior correspondances with Dr. Mann once again go back to [ban ]inflammatory reaction being THE initiator in the development of acne with us as individuals having various triggers for this event.

Hautarzt. 2008 May 18.

[Acne : Current pathophysiologic considerations.][Article in German]

Degitz K, Ochsendorf F.

Dermatologische Gemeinschaftspraxis, Pasinger Bahnhofsplatz1, 81241, München, Deutschland, [email protected]

Seborrhea, follicular hyperkeratosis, propionibacteria, and inflammatory reactions are the most important factors leading to acne. The combination of increased sebum producation and follicular hyperkeratosis facilitates an increased growth of Propionibacterium acnes. Its metabolic products lead to follicular inflammation and, in extreme cases, even to perifollicular abscesses. Sebum production is influenced by androgens, so that abnormalities in androgen levels can produce seborrhea and acne. Follicular hyperkeratosis may be triggered by a relative deficiency in linoleic acid, peroxides from sebum components, and especially by inflammatory mediators such as interleukin-1. Bacterial metabolic products such as lipases, proteases, or chemotactic factors lead to the perifollicular inflammation. This inflammation is not only a response to other pathogenetic factors, but also a cause of acne. An initial mild perifollicular infammation can induce comedogenesis via a variety of mediators. The influence of dietary factors on the initiation and course of acne has recently received increased recognition. A connection has been postulated between acne and a high nutrients with glycemic index, as well as with milk products.

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do you need to keep your total gylcemic load under 80?

are almonds safe , mostly mono fat and low in omega 6?

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do you need to keep your total gylcemic load under 80?

are almonds safe , mostly mono fat and low in omega 6?

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ok just found out dr. mann says keep gycemic load under 70.

avoid high gylcemic cards and you can eat 2-3 servings of whoile graisn a dfay

also he says almonds are safe.

he just did another study showing that high glycemic changes sebum compostion but didnt find it yet.

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RESULTS: After adjusting for age at baseline, height, and energy intake, the multivariate prevalence ratios (95% confidence interval; P value for test of trend) for acne comparing highest (>2 servings/d) with lowest (<1/wk) intake categories in 1996 were 1.16 (1.01, 1.34; 0.77) for total milk, 1.10 (0.94, 1.28; 0.83) for whole/2% milk, 1.17 (0.99, 1.39; 0.08) for low-fat (1%) milk, and 1.19 (1.01, 1.40; 0.02) for skim milk.

After accounting for age at baseline, height and energy intake, the multivariate PRs (95 % CI; p-value for test of trend) for acne comparing highest (2 or more servings per day) to lowest (<1 per week) intake categories in 1996, were 1.20 (1.09, 1.31; <0.001) for total milk, 1.19 (1.06, 1.32; <0.001) for whole milk, 1.17 (1.04, 1.31; 0.002) for low fat milk and 1.19 (1.08, 1.31; <0.001) for skim milk

What do these mean???? I dont get the numbers. Can anybody please explain to me...

Also for the Gl studies: isnt it that theres not really that much correlation between the results. Some showed increase , most decrease but not significant, and only few a significant decrease right?

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