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Unlike glucose, fructose is almost entirely metabolized in the liver. "When fructose reaches the liver," says Dr. William J. Whelan, a biochemist at the University of Miami School of Medicine, "the liver goes bananas and stops everything else to metabolize the fructose." Eating fructose as compared to glucose results in lower circulating insulin levels, leptin, and ghrelin levels postprandially.[17] These hormones are implicated in the control of appetite and satiety, and it is hypothesized that eating lots of fructose could increase the likelihood of weight gain.[18]
Liver cleanse/fush -> helps acne? Therefore a congested liver would constribute to acne. A high fructose diet (many fruits and other foods with fructose or HFCS) will congest your liver.
QUOTE
Fructose also chelates minerals in the blood. This effect is especially important with micronutrients such as copper, chromium and zinc. Since these solutes are normally present in small quantities, chelation of small numbers of ions may lead to deficiency diseases, immune system impairment and even insulin resistance, a component of type II diabetes.[13]
How interesting. Zinc alone has cleared a lot of peoples acne on these forums. Were these people possibly deficient in zinc because of a high fructose diet?
Check out a symptom of deficieny in zinc;
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Signs of zinc deficiency include hair loss, skin lesions, diarrhea, and wasting of body tissues. Eyesight, taste, smell and memory are also connected with zinc. A deficiency in zinc can cause malfunctions of these organs and functions.
Skin lesions? Interesting. Malfunctions with the skin organ and it's function? Even more interesting!
Perhaps the most startling thing I have learned today about fructose:
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There is a concern with Diabetic 1 patients and the apparent low GI of fructose. Fructose gives as high blood sugar spike as that obtained with glucose. In fact, GI only applies to high starch foods. The basic GI definition is chemically incorrect. This is because the body blood glucose response is "standardized" with 50g of glucose, while the GI Researchers use 50g of digestible carbohydrate as a reference quantity.
It's interesting that many of our knowlegable members and veterans on these boards have said that spikes in blood sugar can indirectly cause the keratinization of pores.
Please, do more reasearch of your own and post what you think!
The following information was taken from http://en.wikipedia.org/wiki/Fructose
Using the following sources of information;
1^ Buchs, AE; Sasson S, Joost HG, Cerasi E. (1998). "Characterization of GLUT5 domains responsible for fructose transport". Endocrinology 139: 827-31. PMID 12399260.
2^ Elliott SS, Keim NL, Stern JS, Teff K, Havel PJ (2002). "Fructose, weight gain, and the insulin resistance syndrome". Am. J. Clin. Nutr. 76 (5): 911��“22. PMID 12399260.
3^ Lustig RH (2006). "Childhood obesity: behavioral aberration or biochemical drive? Reinterpreting the First Law of Thermodynamics". Nature clinical practice. Endocrinology & metabolism 2 (8): 447��“58. doi:10.1038/ncpendmet0220. PMID 16932334.
4^ Isganaitis E, Lustig RH (2005). "Fast food, central nervous system insulin resistance, and obesity". Arterioscler. Thromb. Vasc. Biol. 25 (12): 2451��“62. doi:10.1161/01.ATV.0000186208.06964.91. PMID 16166564.
5^ Hughes TA, Atchison J, Hazelrig JB, Boshell BR (1989). "Glycemic responses in insulin-dependent diabetic patients: effect of food composition". Am. J. Clin. Nutr. 49 (4): 658��“66. PMID 2929488.
6^ Wylie-Rosett, Judith; et al. (2004). "Carbohydrates and Increases in Obesity: Does the Type of Carbohydrate Make a Difference?". Obesity Res 12: 124S-129S.
7^ a b Havel PJ (2001). "Peripheral signals conveying metabolic information to the brain: short-term and long-term regulation of food intake and energy homeostasis". Exp. Biol. Med. (Maywood) 226 (11): 963��“77. PMID 11743131.
8^ Dennison BA, Rockwell HL, Baker SL (1997). "Excess fruit juice consumption by preschool-aged children is associated with short stature and obesity". Pediatrics 99 (1): 15��“22. PMID 8989331.
9^ J�rgens H, Haass W, Casta�eda TR, et al (2005). "Consuming fructose-sweetened beverages increases body adiposity in mice". Obes. Res. 13 (7): 1146��“56. PMID 16076983.
10^ Bantle JP, Raatz SK, Thomas W, Georgopoulos A (2000). "Effects of dietary fructose on plasma lipids in healthy subjects". Am. J. Clin. Nutr. 72 (5): 1128��“34. PMID 11063439.
11^ http://www.enerex.ca/articles/whey_protein_and_fructose.htm
12^ Melanson, K.; et al. (2006). "Eating Rate and Satiation.". Obesity Society (NAASO) 2006 Annual Meeting, October 20-24,Hynes Convention Center, Boston, Massachusett..
13^ Higdon, J. (2003). "Chromium". Linus Pauling Institute, Oregon State U..
14^ Field, Meira (Fall 2001). "Wise Traditions in Food, Farming and the Healing Arts". Weston A. Price Foundation.
15^ McPherson, JD; Shilton BH, Walton DJ (November 1988). "Role of fructose in glycation and cross-linking of proteins. PMID 3132203". Biochemistry 27 (5): 1901-7.
16^ Levi, B; Werman MJ (1998). "Fulltext Long-term fructose consumption accelerates glycation and several age-related variables in male rats. PMID 9732303". J Nutr 128: 1442-9.
17^ Teff, KL; Elliott SS, Tsch�p M, Kieffer TJ, Rader D, Heiman M, Townsend RR, Keim NL, D'Alessio D, Havel PJ (June 2004). "Fulltext Dietary fructose reduces circulating insulin and leptin, attenuates postprandial suppression of ghrelin, and increases triglycerides in women. PMID 15181085". J Clin Endocrinol Metab. 89 (6): 2963-72.
18^ Swan, Norman. ABC Radio National, The Health Report, The Obesity Epidemic. Retrieved on 2007-07-15.
