An article released this month in the British Journal of Dermatology took a look at the evidence scientists have boldly gathered from 1950 through 2010 regarding when and where acne bacteria tends to exist and in what amounts. The patterns they found are intriguing and seem to suggest that acne bacteria, known as P. Acnes, may not cause clogged pores.
Over the past 60 years, various researchers have taken biopsies of “normal” skin, acne prone skin, and inflamed and noninflamed acne lesions, and then counted the bacteria they found. It turns out that across the vast majority of the 14 studies of this nature, acne bacteria is not always present, even in active acne lesions. A small but compelling percentage of acne lesions are “sterile” and contain no bacteria whatsoever.
Faced with this kind of consistent evidence, the authors hypothesize that acne bacteria does not initiate acne. How could it, they seem to ask us to ponder, if it doesn’t exist in all acne lesions?
However, the authors go on to state that after a pore has become clogged, acne bacteria can make the situation worse through a number of means, including increasing the skin’s cell production, causing stickiness inside the pore, and kicking inflammation into higher gear, amongst others.
So what causes acne? Is it our body’s immune response? Inflammation? Genetics? Vitamin/Mineral deficiency? The search continues…but as we move forward, let’s keep this evidence regarding bacteria in mind.